K.‐H. Sontag - Concepedia

Author

K.‐H. Sontag

Max Planck Institute of Experimental Medicine

Also Known As

K H Sontag, K-H Sontag, K. -H. Sontag, K. H. Sontag, K.-H. Sontag, K.H. Sontag, K.‐H. Sontag

Publications

1.2K

Citations

H-Index

Concepts

All Affiliations

Max Planck Institute of Experimental Medicine University of Göttingen Radboud University Nijmegen Polish Academy of Sciences Maj Institute of Pharmacology

About

K.‐H. Sontag is an author at Max Planck Institute of Experimental Medicine specializing in medicine, neuroscience, and neuropharmacology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	20	25	905
2	Health Sciences	10	10	330
3	Social Sciences	10	10	399
4	Engineering	1	1	17
5	Natural Sciences	1	1	19

K.‐H. Sontag

Publications

1.1K

Citations

H-Index

Page 1

	Year	Citations
Morphine-induced muscular rigidity in rats P. Wand, K. Kuschinsky, K.‐H. Sontag European Journal of Pharmacology MechanobiologyHealth SciencesNeuroanatomyPain MedicinePhysiology	1973	125
Caudate nucleus and programming behaviour in cats: Role of dopamine in switching motor patterns R.M.A. Jaspers, Michael Schwarz, K.‐H. Sontag, Behavioural Brain Research Neurobiological MechanismCognitive ScienceNeural MechanismBehavioral NeuroscienceNeuroanatomy	1984	88
Muscular rigidity and delineation of a dopamine-specific neostriatal subregion: Tonic EMG activity in rats Bart Ellenbroek, Michael Schwarz, K.‐H. Sontag, Brain Research Neurobiological MechanismDopamine-specific Neostriatal SubregionBrain StructureNeurophysiologyNeuroanatomy	1985	87
Sensory‐motor processing in substantia nigra pars reticulata in conscious cats. Michael Schwarz, K.‐H. Sontag, P. Wand The Journal of Physiology	1984	63
Retinal ischemia induced by occlusion of both common carotid arteries in rats as demonstrated by electroretinography F. Block, Martin Schwarz, K.‐H. Sontag Neuroscience Letters Ocular DiseaseRetinaOphthalmologyCardiovascular DiseaseMedicine	1992	62
Substantia nigra as a station that not only transmits, but also transforms, incoming signals for its behavioural expression: Striatal dopamine and GABA-mediated responses of pars reticulata neurons A.R. Cools, R.M.A. Jaspers, Wacław Kolasiewicz, Behavioural Brain Research Striatal DopamineBehavioural ExpressionNeurobiological MechanismBehavioral NeuroscienceNeuroanatomy	1983	51
The glia‐derived protease nexin 1 persists for over 1 year in rat brain areas selectively lesioned by transient global ischaemia Cordula Nitsch, Alessandra L. Scotti, Denis Monard, European Journal of Neuroscience	1993	43
Posttreatment with EPC-K1, an inhibitor of lipid peroxidation and of phospholipase A2 activity, reduces functional deficits after global ischemia in rats F. Block, M.E. Kunkel, K.‐H. Sontag Brain Research Bulletin InflammationMetabolic SyndromeOxysterolBiochemistryGlobal Ischemia	1995	38
Rigidity and catalepsy after injections of muscimol into the ventromedial thalamic nucleus: an electromyographic study in the rat Thomas Klockgether, Michael Schwarz, L. Turski, Experimental Brain Research Ventromedial Thalamic NucleusElectromyographic StudyVestibular SystemNeurophysiologyNeuroanatomy	1985	36
Damage of substantia nigra pars reticulata during pilocarpine-induced status epilepticus in the rat: immunohistochemical study of neurons, astrocytes and serum-protein extravasation Rainald Schmidt‐Kastner, Christine Heim, K.‐H. Sontag Experimental Brain Research Pilocarpine-induced Status EpilepticusNeurophysiologyNeuroanatomyMedicineNeuropharmacology	1991	35

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