Thomas J. Ferro - Concepedia

Author

Thomas J. Ferro

Albany Medical Center Hospital

Also Known As

Ferro, Thomas, T Ferro, T J Ferro, T. Ferro, T. J. Ferro, Thomas Ferro, Thomas J Ferro, Thomas J. Ferro, Tomás Ferro

Publications

2.1K

Citations

H-Index

Concepts

All Affiliations

Albany Medical Center Hospital Albany Stratton VA Medical Center Albany United States Department of Veterans Affairs Veterans Health Administration Virginia Commonwealth University

About

Thomas J. Ferro is an author at Albany Medical Center Hospital specializing in medicine, inflammation, and vascular biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	17	22	1.2K
2	Natural Sciences	4	4	383
3	Public Health	2	2	25
4	Allergy	2	2	63
5	Health Sciences	1	1	29

Thomas J. Ferro

Publications

1.6K

Citations

H-Index

Page 1

	Year	Citations
Increased endothelial albumin permeability mediated by protein kinase C activation. Joseph J. Lynch, Thomas J. Ferro, F. A. Blumenstock, Journal of Clinical Investigation 125I-albumin PermeabilityCell AdhesionCellular PhysiologyInflammationAngiogenesis	1990	270
Recombinant tumor necrosis factor increases pulmonary vascular permeability independent of neutrophils. Christopher Horvath, Thomas J. Ferro, Gary Jesmok, Proceedings of the National Academy of Sciences Inflammatory Lung DiseaseLung InflammationIncreased PermeabilityImmunologyPathology	1988	153
Mechanisms of pulmonary edema induced by tumor necrosis factor-alpha. Denise C. Hocking, Patricia G. Phillips, Thomas J. Ferro, Circulation Research Acute Lung InjuryInflammatory Lung DiseaseLung InflammationImmunologyOxidative Stress	1990	117
TNF-alpha induces peroxynitrite-mediated depletion of lung endothelial glutathione via protein kinase C D. T. Phelps, Thomas J. Ferro, P. J. Higgins, American Journal of Physiology-Lung Cellular and Molecular Physiology Inflammatory Lung DiseaseLung InflammationLipid PeroxidationImmunologyCell Death	1995	117
Protein kinase C-α mediates endothelial barrier dysfunction induced by TNF-α Thomas J. Ferro, Paul Neumann, Nancy Gertzberg, American Journal of Physiology-Lung Cellular and Molecular Physiology	2000	117
Protein kinase inhibitor prevents pulmonary edema in response to H2O2 A. Johnson, Patricia G. Phillips, Denise C. Hocking, American Journal of Physiology-Heart and Circulatory Physiology Acute Lung InjuryLung InflammationLung WeightOxidative StressInflammation	1989	82
Historical cohort study examining comparative effectiveness of albuterol inhalers with and without integrated dose counter for patients with asthma or chronic obstructive pulmonary disease David Price, Anna Rigazio, Mary Buatti Small, Journal of Asthma and Allergy	2016	66
Alveolar macrophages, blood monocytes, and density-fractionated alveolar macrophages differ in their ability to promote lymphocyte proliferation to mitogen and antigen. Thomas J. Ferro, Jeffrey A. Kern, Jack A. Elias, PubMed T-regulatory CellImmunologyImmunologic MechanismImmune SystemImmunotherapy	1987	52
Endothelial barrier dysfunction and p42 oxidation induced by TNF-alpha are mediated by nitric oxide Thomas J. Ferro, Nancy Gertzberg, Lynn A. Selden, American Journal of Physiology-Lung Cellular and Molecular Physiology Nitric OxideLipid PeroxidationImmunologyCytoskeletonRedox Biology	1997	49
Fluticasone propionate/salmeterol 250/50 μg versus salmeterol 50 μg after chronic obstructive pulmonary disease exacerbation Jill Ohar, Glenn Crater, Amanda Emmett, Respiratory Research	2014	48

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