John H.B. Bridge - Concepedia

Author

John H.B. Bridge

Also Known As

J.H. Bridge, JOHN H B BRIDGE, JOHN H. B. BRIDGE, John Bridge, John H Bridge, John H. B. Bridge, John H. Bridge, John H.B. Bridge, John HB Bridge, John H.B. Bridge

Publications

2.9K

Citations

H-Index

Concepts

All Affiliations

University of Utah University of California, Los Angeles Johns Hopkins University Johns Hopkins Medicine University of Auckland

About

John H.B. Bridge is an author at University of Utah specializing in medicine, cellular physiology, and cardiovascular physiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	18	20	1.4K
2	Engineering	4	4	180
3	Social Sciences	2	2	130
4	Health Sciences	1	1	272
5	Anatomy	1	1	85

John H.B. Bridge

Publications

2.4K

Citations

H-Index

Page 1

	Year	Citations
The Relationship Between Charge Movements Associated with <i>I</i> <sub>Ca</sub> and <i>I</i> <sub>Na-Ca</sub> in Cardiac Myocyte John H.B. Bridge, John R. Smolley, Kenneth W. Spitzer Science Cardiac MuscleCardiac MyocyteCellular PhysiologyMuscle PhysiologyElectrophysiological Evaluation	1990	272
Effects of Overexpression of the Na <sup>+</sup> -Ca <sup>2+</sup> Exchanger on [Ca <sup>2+</sup> ] <sub>i</sub> Transients in Murine Ventricular Myocytes Atsushi Yao, Zhi Su, Akihiko Nonaka, Circulation Research Cardiac MuscleNeurotransmissionMurine Ventricular MyocytesCellular PhysiologyElectrolyte Disturbance	1998	200
Na-Ca Exchange and the Trigger for Sarcoplasmic Reticulum Ca Release: Studies in Adult Rabbit Ventricular Myocytes Sheldon E. Litwin, Jun Li, John H.B. Bridge Biophysical Journal Cardiac MuscleCardiomyopathyMolecular PhysiologyPhysiologyCardiology	1998	170
Dyssynchronous Ca <sup>2+</sup> Sparks in Myocytes From Infarcted Hearts Sheldon E. Litwin, Dongfang Zhang, John H.B. Bridge Circulation Research	2000	169
Enhanced Na <sup>+</sup> -Ca <sup>2+</sup> Exchange in the Infarcted Heart Sheldon E. Litwin, John H.B. Bridge Circulation Research	1997	159
Subcellular Structures and Function of Myocytes Impaired During Heart Failure Are Restored by Cardiac Resynchronization Therapy Frank B. Sachse, Natalia S. Torres, Eleonora Savio‐Galimberti, Circulation Research	2012	130
Properties of Ca<sup>2+</sup> sparks evoked by action potentials in mouse ventricular myocytes John H.B. Bridge, Philip R. Ershler, Mark B. Cannell The Journal of Physiology	1999	117
Relaxation of Isolated Ventricular Cardiomyocytes by a Voltage-Dependent Process John H.B. Bridge, Kenneth W. Spitzer, Philip R. Ershler Science	1988	108
Activation of reverse Na<sup>+</sup>-Ca<sup>2+</sup>exchange by the Na<sup>+</sup>current augments the cardiac Ca<sup>2+</sup>transient: evidence from NCX knockout mice Robert Larbig, Natalia S. Torres, John H.B. Bridge, The Journal of Physiology Cardiac MuscleReverse NaSynaptic TransmissionMurine HeartSynaptic Signaling	2010	93
Abnormal myocyte Ca<sup>2+</sup>homeostasis in rabbits with pacing-induced heart failure Atsushi Yao, Zhi Su, Akihiko Nonaka, American Journal of Physiology-Heart and Circulatory Physiology	1998	87

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