Concepedia

Author

Banishree Saha

University of Massachusetts Chan Medical School

Also Known As

Banishree Saha

37

Publications

2.9K

Citations

25

H-Index

49

Concepts

All Affiliations

University of Massachusetts Chan Medical SchoolIndian Institute of Science BangaloreUniversity of Massachusetts Amherst
BS

About

Banishree Saha is an author at University of Massachusetts Chan Medical School specializing in medicine, immunology, and cell biology.

Top Concepts

#ConceptH-IndexPublicationsCitations

1

Medicine

8

8

1.1K

2

Cell Biology

5

5

466

3

Health Sciences

1

1

163

4

Natural Sciences

1

1

92

5

Allergy

1

1

52

Banishree Saha

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23

Publications

2.5K

Citations

22

H-Index

YearCitations

Increased number of circulating exosomes and their microRNA cargos are potential novel biomarkers in alcoholic hepatitis

Fatemeh Momen‐Heravi, Banishree Saha, Karen Kodys,

Journal of Translational Medicine

2015

297

The pro-inflammatory effects of miR-155 promote liver fibrosis and alcohol-induced steatohepatitis

Shashi Bala, Tímea Csák, Banishree Saha,

Journal of Hepatology

2016

296

MicroRNA Cargo of Extracellular Vesicles from Alcohol-exposed Monocytes Signals Naive Monocytes to Differentiate into M2 Macrophages

Banishree Saha, Fatemeh Momen‐Heravi, Karen Kodys,

Journal of Biological Chemistry

2015

223

Pharmacological Inhibition of CCR2/5 Signaling Prevents and Reverses Alcohol‐Induced Liver Damage, Steatosis, and Inflammation in Mice

Aditya Ambade, Patrick Lowe, Karen Kodys,

Hepatology

2018

213

Abnormal neutrophil traps and impaired efferocytosis contribute to liver injury and sepsis severity after binge alcohol use

Terence N. Bukong, Yeonhee Cho, Arvin Iracheta‐Vellve,

Journal of Hepatology

2018

163

Metabolic danger signals, uric acid and ATP, mediate inflammatory cross-talk between hepatocytes and immune cells in alcoholic liver disease

Jan Petrášek, Arvin Iracheta‐Vellve, Banishree Saha,

Journal of Leukocyte Biology

2015

150

Progression of non-alcoholic steatosis to steatohepatitis and fibrosis parallels cumulative accumulation of danger signals that promote inflammation and liver tumors in a high fat–cholesterol–sugar diet model in mice

Michal Ganz, Terence N. Bukong, Tímea Csák,

Journal of Translational Medicine

2015

137

Inhibition of sterile danger signals, uric acid and ATP, prevents inflammasome activation and protects from alcoholic steatohepatitis in mice

Arvin Iracheta‐Vellve, Jan Petrášek, Abhishek Satishchandran,

Journal of Hepatology

2015

135

Extracellular vesicles from mice with alcoholic liver disease carry a distinct protein cargo and induce macrophage activation through heat shock protein 90

Banishree Saha, Fatemeh Momen‐Heravi, István Fűri,

Hepatology

2017

122

Hepatocellular carcinoma is accelerated by NASH involving M2 macrophage polarization mediated by hif-1<b>α</b>induced IL-10

Aditya Ambade, Abhishek Satishchandran, Banishree Saha,

OncoImmunology

2016

96

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