Prosenjit Saha - Concepedia

Author

Prosenjit Saha

Chittaranjan National Cancer Institute ORCID

Also Known As

P Saha, PROSENJIT SAHA, Prosenjit Saha, Sabato, G, Saha Prosenjit

Publications

Citations

H-Index

Concepts

All Affiliations

Chittaranjan National Cancer Institute West Virginia University West Virginia University Hospitals University of Calcutta Medical College and Hospital, Kolkata

About

Prosenjit Saha is an author at Chittaranjan National Cancer Institute specializing in medicine, pharmacology, and cell biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	18	21	677
2	Biochemistry	5	5	186
3	Health Sciences	3	3	167
4	Natural Sciences	3	3	77
5	Allergy	3	3	131

Prosenjit Saha

Publications

938

Citations

H-Index

Page 1

	Year	Citations
Evaluation of the anticarcinogenic activity of <i>Swertia chirata</i> Buch.Ham, an Indian medicinal plant, on DMBA‐induced mouse skin carcinogenesis model Prosenjit Saha, Suvra Mandal, Ashes Das, Phytotherapy Research Nutraceutical IngredientSwertia Chirata Buch.hamCell ProliferationDermatologyAnticarcinogenic Activity	2004	81
Black tea polyphenols suppress cell proliferation and induce apoptosis during benzo(a)pyrene-induced lung carcinogenesis Soumya Banerjee, Soumen K. Manna, Prosenjit Saha, European Journal of Cancer Prevention Chemoprevention StrategyPyrene-induced Lung CarcinogenesisPathologyCell DeathCell Proliferation	2005	58
Tea polyphenols EGCG and TF restrict tongue and liver carcinogenesis simultaneously induced by N-nitrosodiethylamine in mice Subhayan Sur, Debolina Pal, Atish Barua, Toxicology and Applied Pharmacology Chemoprevention StrategyOncogenic AgentMedicinePathologyLiver Carcinogenesis	2016	57
Amarogentin can reduce hyperproliferation by downregulation of Cox-II and upregulation of apoptosis in mouse skin carcinogenesis model Prosenjit Saha, Suvra Mandal, Ashes Das, Cancer Letters Oncogenic AgentPhotocarcinogenesisApoptosisCell DeathTumor Suppressor	2006	50
Regulation of hazardous exposure by protective exposure: Modulation of phase II detoxification and lipid peroxidation byCamellia sinensis andSwertia chirata Prosenjit Saha, Sukta Das Teratogenesis Carcinogenesis and Mutagenesis	2003	39
Pongapin and Karanjin, furanoflavanoids of <scp><i>Pongamia pinnata</i></scp>, induce G2/M arrest and apoptosis in cervical cancer cells by differential reactive oxygen species modulation, DNA damage, and nuclear factor kappa‐light‐chain‐enhancer of activated B cell signaling Debolina Pal, Subhayan Sur, Suvra Mandal, Phytotherapy Research Chemoprevention StrategyG2/m ArrestApoptosisCell DeathCancer Biology	2019	38
Deregulation of the CD44-NANOG-MDR1 associated chemoresistance pathways of breast cancer stem cells potentiates the anti-cancer effect of Kaempferol in synergism with Verapamil Sourav Kumar Nandi, Tanaya Roychowdhury, Samit Chattopadhyay, Toxicology and Applied Pharmacology MedicinePharmacologyBreast CancerAnti-cancer EffectChemoresistance Pathways	2022	38
Indole-3-Carbinol (I3C) enhances the sensitivity of murine breast adenocarcinoma cells to doxorubicin (DOX) through inhibition of NF-κβ, blocking angiogenesis and regulation of mitochondrial apoptotic pathway Subhadip Hajra, Arup Ranjan Patra, Abhishek Basu, Chemico-Biological Interactions	2018	37
Exosome-sheathed porous silica nanoparticle-mediated co-delivery of 3,3′-diindolylmethane and doxorubicin attenuates cancer stem cell-driven EMT in triple negative breast cancer Rupali Sarkar, Souradeep Biswas, Rituparna Ghosh, Journal of Nanobiotechnology	2024	33
Role of Sp1 and HNF1 transcription factors in SGLT1 regulation during chronic intestinal inflammation Ramesh Kekuda, Prosenjit Saha, Uma Sundaram American Journal of Physiology-Gastrointestinal and Liver Physiology Chronic Inflammatory DiseasesImmunologyGene Regulatory NetworkChronic Intestinal InflammationInflammation	2008	32

Page 1