Concepedia

Author

Thomas M. Ashton

University of Oxford

Also Known As

Ashton, Thomas M., T Ashton, T. Ashton, Thomas Ashton, Thomas M Ashton, Thomas M. Ashton

17

Publications

1.7K

Citations

9

H-Index

0

Concepts

All Affiliations

University of OxfordCRUK/MRC Oxford Institute for Radiation OncologyJohn Radcliffe HospitalUniversity of Copenhagen
TM

About

Thomas M. Ashton is an author at University of Oxford.

Thomas M. Ashton

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6

Publications

462

Citations

6

H-Index

YearCitations

The anti-malarial atovaquone increases radiosensitivity by alleviating tumour hypoxia

Thomas M. Ashton, Emmanouil Fokas, Leoni A. Kunz‐Schughart,

Nature Communications

2016

245

Pathways for Holliday Junction Processing during Homologous Recombination in <i>Saccharomyces cerevisiae</i>

Thomas M. Ashton, Hocine W Mankouri, A Heidenblut,

Molecular and Cellular Biology

2011

62

Holliday junction-containing DNA structures persist in cells lacking Sgs1 or Top3 following exposure to DNA damage

Hocine W Mankouri, Thomas M. Ashton, Ian D. Hickson

Proceedings of the National Academy of Sciences

2011

56

Regulation of O2 consumption by the PI3K and mTOR pathways contributes to tumor hypoxia

Kamila Hussien, Emmanouil Fokas, Pavitra Kannan,

Radiotherapy and Oncology

2014

49

TOPK modulates tumour-specific radiosensitivity and correlates with recurrence after prostate radiotherapy

Giacomo Pirovano, Thomas M. Ashton, Katharine Herbert,

British Journal of Cancer

2017

26

The anti-malarial drug atovaquone potentiates platinum-mediated cancer cell death by increasing oxidative stress

James T. Coates, Gonzalo Rodríguez‐Berriguete, Rathi Puliyadi,

Cell Death Discovery

2020

24

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