M.A. Christine Pratt

Author

Also Known As

M A C Pratt, M A Christine Pratt, M A Pratt, M AC Pratt, M. A. C. Pratt, M. A. Christine Pratt, M.A. Christine Pratt, M.A.C. Pratt, M.A.Christine Pratt, M.A. Christine Pratt

Publications

1.7K

Citations

H-Index

Concepts

All Affiliations

University of Ottawa Carleton University Ottawa Hospital University of Illinois Urbana-Champaign University of Toronto

About

M.A. Christine Pratt is an author at University of Ottawa specializing in medicine, cell biology, and tumor biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	16	16	1.2K
2	Cell Biology	14	14	1.1K
3	Immunology	3	3	190
4	Endocrinology	2	2	125
5	Health Sciences	1	1	19

M.A. Christine Pratt

Publications

1.6K

Citations

H-Index

Page 1

	Year	Citations
Estrogen promotes chemotherapeutic drug resistance by a mechanism involving Bcl-2 proto-oncogene expression in human breast cancer cells. Christine Teixeira, John C. Reed, M.A. Christine Pratt PubMed Breast OncologyChemoprevention StrategyCell DeathCancer BiologyTumor Biology	1995	421
A dominant negative mutation of the alpha retinoic acid receptor gene in a retinoic acid-nonresponsive embryonal carcinoma cell. M.A. Christine Pratt, Jarmila Králová, Michael W. McBurney Molecular and Cellular Biology Mutant GeneP19 CellsDevelopmental BiologyCell RegulationOncogenic Agent	1990	131
Persistent Activation of NF-κB in BRCA1-Deficient Mammary Progenitors Drives Aberrant Proliferation and Accumulation of DNA Damage A. C. SAU, Rosanna Lau, Miguel A. Cabrita, Cell stem cell Genome InstabilityDna DamageCell RegulationAberrant ProliferationImmune Cell Development	2016	113
Estrogen Withdrawal-Induced NF-κB Activity and Bcl-3 Expression in Breast Cancer Cells: Roles in Growth and Hormone Independence M.A. Christine Pratt, Tanya E. Bishop, Dawn White, Molecular and Cellular Biology Breast OncologyHormone IndependenceGynecologyP50 DimersMammary Gland Development	2003	107
CDK2 Is a Target for Retinoic Acid-Mediated Growth Inhibition in MCF-7 Human Breast Cancer Cells Christine Teixeira, M.A. Christine Pratt Molecular Endocrinology	1997	90
The canonical NF-κB pathway is required for formation of luminal mammary neoplasias and is activated in the mammary progenitor population M.A. Christine Pratt, Emma Tibbo, S. Robertson, Oncogene Mammary Progenitor PopulationMedicineCanonical Nf-κb PathwayCancer GrowthCancer Cell Biology	2009	78
NF‐κB and estrogen receptor α interactions: Differential function in estrogen receptor‐negative and ‐positive hormone‐independent breast cancer cells Nathalie Gionet, Deidre Jansson, Sylvie Mader, Journal of Cellular Biochemistry	2009	52
NF-κB-Dependent Role for Cold-Inducible RNA Binding Protein in Regulating Interleukin 1β Christian Brochu, Miguel A. Cabrita, Brian D. Melanson, PLoS ONE Molecular RegulationImmunologyImmune RegulationOligonucleotide MicroarraysInnate Immunity	2013	51
Preferential Estrogen Receptor β Ligands Reduce Bcl-2 Expression in Hormone-Resistant Breast Cancer Cells to Increase Autophagy Samantha C. Ruddy, Rosanna Lau, Miguel A. Cabrita, Molecular Cancer Therapeutics	2014	47
Estrogen activates raf-1 kinase and induces expression of Egr-1 in MCF-7 breast cancer cells M.A. Christine Pratt, Abheha Satkunaratnam, Denise M. Novosad Molecular and Cellular Biochemistry	1998	45

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