Histone acetylation by CBP and p300 at double-strand break sites facilitates SWI/SNF chromatin remodeling and the recruitment of non-homologous end joining factors

Hideaki Ogiwara, Ayako Ui, Atsushi Otsuka, +6

Oncogene

Histone ModificationsChromatinGenome InstabilityChromatin RemodelingEpigenetic Change +7

The ACF1 Complex Is Required for DNA Double-Strand Break Repair in Human Cells

Li Lan, Ayako Ui, Satoshi Nakajima, +8

Molecular Cell

ChromatinGenome InstabilityDna DamageChromatin RemodelingHuman Cells +5

SWI/SNF Factors Required for Cellular Resistance to DNA Damage Include ARID1A and ARID1B and Show Interdependent Protein Stability

Reiko Watanabe, Ayako Ui, Shin‐ichiro Kanno, +4

Cancer Research

Transcriptional Elongation Factor ENL Phosphorylated by ATM Recruits Polycomb and Switches Off Transcription for DSB Repair

Ayako Ui, Yuko Nagaura, Akira Yasui

Molecular Cell

ChromatinTranscriptional RegulationAtm Recruits PolycombSwitches Off TranscriptionNatural Sciences +7

Fine-Tuning of DNA Damage-Dependent Ubiquitination by OTUB2 Supports the DNA Repair Pathway Choice

Kiyoko Kato, Kazuhiro Nakajima, Ayako Ui, +3

Molecular Cell

ChromatinGenome InstabilityGenomic MechanismMolecular BiologyDna Damage-dependent Ubiquitination +3

Curcumin suppresses multiple DNA damage response pathways and has potency as a sensitizer to PARP inhibitor

Hideaki Ogiwara, Ayako Ui, Bunsyo Shiotani, +3

Carcinogenesis

The N-terminal region of Sgs1, which interacts with Top3, is required for complementation of MMS sensitivity and suppression of hyper-recombination in sgs1 disruptants

Ayako Ui, Y. Satoh, Fumitoshi Onoda, +3

Molecular Genetics and Genomics

Sgs1 DisruptantsGenome InstabilitySignal TransductionCell RegulationMolecular Biology +6

SMC6 is required for MMS-induced interchromosomal and sister chromatid recombinations in Saccharomyces cerevisiae

Fumitoshi Onoda, Masahiro Takeda, Masayuki Seki, +5

DNA repair

Mms-induced InterchromosomalChromatinGenome InstabilitySister Chromatid RecombinationsChromatin Structure +11

Possible involvement of LKB1-AMPK signaling in non-homologous end joining

Ayako Ui, Hideaki Ogiwara, Shuro Nakajima, +8

Oncogene

Protein FunctionSignal TransductionMolecular PhysiologySignaling PathwayNon-homologous End Joining +4

The ability of Sgs1 to interact with DNA topoisomerase III is essential for damage-induced recombination

Ayako Ui, Masayuki Seki, Hideaki Ogiwara, +4

DNA repair

ChromatinGenome InstabilityDna DamageChromatin RemodelingDna Topoisomerase Iii +5