Carl Y. Saab - Concepedia

Author

Carl Y. Saab

Rhode Island Hospital ORCID

Also Known As

C Saab, C. Saab, C. Y. Saab, C.Y. Saab, Carl Saab, Carl Y Saab, Carl Y. Saab, Carl saab, Joshua Klein, Laxmi Parida

Publications

3.5K

Citations

H-Index

Concepts

All Affiliations

Rhode Island Hospital Brown University Yale University Providence College VA Connecticut Healthcare System

About

Carl Y. Saab is an author at Rhode Island Hospital specializing in medicine, neuroscience, and neurophysiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	21	25	1.8K
2	Health Sciences	14	15	1K
3	Social Sciences	9	9	668
4	Engineering	1	1	35
5	Natural Sciences	1	1	18

Carl Y. Saab

Publications

2.6K

Citations

H-Index

Page 1

	Year	Citations
Upregulation of Sodium Channel Na<sub>v</sub>1.3 and Functional Involvement in Neuronal Hyperexcitability Associated with Central Neuropathic Pain after Spinal Cord Injury Bryan C. Hains, Joshua P. Klein, Carl Y. Saab, Journal of Neuroscience Central Neuropathic PainPeripheral Nerve InjuryPain MedicineNeuropathic PainMolecular Pain	2003	362
Altered Sodium Channel Expression in Second-Order Spinal Sensory Neurons Contributes to Pain after Peripheral Nerve Injury Bryan C. Hains, Carl Y. Saab, Joshua P. Klein, Journal of Neuroscience	2004	270
Phenytoin Protects Spinal Cord Axons and Preserves Axonal Conduction and Neurological Function in a Model of Neuroinflammation In Vivo Albert Lo, Carl Y. Saab, Joel A. Black, Journal of Neurophysiology Cellular NeurobiologySocial SciencesNeuroinflammationNeuroregenerationExperimental Neuropathology	2003	187
Changes in electrophysiological properties and sodium channel Nav1.3 expression in thalamic neurons after spinal cord injury Bryan C. Hains, Carl Y. Saab, Stephen G. Waxman Brain	2005	186
Long-term protection of central axons with phenytoin in monophasic and chronic-relapsing EAE J.A. Black, Shuang Liu, Bryan C. Hains, Brain ImmunologySocial SciencesNeuroinflammationNeuroregenerationExperimental Neuropathology	2006	118
Alterations in Burst Firing of Thalamic VPL Neurons and Reversal by Na<sub>v</sub>1.3 Antisense After Spinal Cord Injury Bryan C. Hains, Carl Y. Saab, Stephen G. Waxman Journal of Neurophysiology Synaptic TransmissionNeurotransmissionCellular NeurobiologySynaptic SignalingSocial Sciences	2006	102
Cortical theta is increased while thalamocortical coherence is decreased in rat models of acute and chronic pain Brian W. LeBlanc, Theresa R. Lii, Andrew E. Silverman, Pain Pain MedicineNeuropathic PainRat ModelPain SyndromeNeurology	2014	93
Sodium channel blockade with phenytoin protects spinal cord axons, enhances axonal conduction, and improves functional motor recovery after contusion SCI Bryan C. Hains, Carl Y. Saab, Albert Lo, Experimental Neurology NeuroregenerationSpinal Cord InjuryPeripheral Nerve InjuryNeurophysiologyMedicine	2004	92
Neutrophils invade lumbar dorsal root ganglia after chronic constriction injury of the sciatic nerve Natalia Quijada Morin, Samuel A. Owolabi, Mark W. Harty, Journal of Neuroimmunology Lumbosacral RadiculopathySpinal Cord InjuryPeripheral Nerve InjuryMedicinePathology	2007	89
Unilateral Focal Burn Injury Is Followed by Long-Lasting Bilateral Allodynia and Neuronal Hyperexcitability in Spinal Cord Dorsal Horn Yu-Wen Chang, Andrew M. Tan, Carl Y. Saab, Journal of Pain Spinal Cord InjuryNeuronal HyperexcitabilitySpinal DisorderNeurophysiologyNeuroanatomy	2009	84

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