Ozan Baytaş

Mutations in mitochondrial enzyme GPT2 cause metabolic dysfunction and neurological disease with developmental and progressive features

Qing Ouyang, Tojo Nakayama, Ozan Baytaş et al. · Proceedings of the National Academy of Sciences · 2016 · 64 citations · Full text

Cellular resilience: 5-HT neurons in Tph2−/− mice retain normal firing behavior despite the lack of brain 5-HT

Alberto Montalbano, Jonas Waider, Mario Barbieri et al. · European Neuropsychopharmacology · 2015 · 23 citations · Full text

Synaptic Plasticity, Brain Development, Cellular Neuroscience +13

Longitudinal MRI findings in patient with <i>SLC25A12</i> pathogenic variants inform disease progression and classification

Brian C. Kavanaugh, Emily B. Warren, Ozan Baytaş et al. · American Journal of Medical Genetics Part A · 2019 · 17 citations · Full text

Disease Progression, Neurological Disorder, Genetics +20

Loss of mitochondrial enzyme GPT2 causes early neurodegeneration in locus coeruleus

Ozan Baytaş, Julie A. Kauer, Eric M. Morrow · Neurobiology of Disease · 2022 · 13 citations · Full text

Mitochondrial enzyme GPT2 regulates metabolic mechanisms required for neuron growth and motor function<i>in vivo</i>

Ozan Baytaş, Shawn M. Davidson, Ralph J. DeBerardinis et al. · Human Molecular Genetics · 2021 · 11 citations · Full text