Mona M. Freidin - Concepedia

Author

Mona M. Freidin

Albert Einstein College of Medicine ORCID

Also Known As

M Freidin, M. Freidin, M. M. Freidin, MONA FREIDIN, Mona Freidin, Mona M. Freidin

Publications

1.5K

Citations

H-Index

Concepts

All Affiliations

Albert Einstein College of Medicine SUNY Downstate Health Sciences University University of Pennsylvania University of Illinois Chicago Yeshiva University

About

Mona M. Freidin is an author at Albert Einstein College of Medicine specializing in medicine, cell biology, and cellular physiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	9	9	694
2	Cell Biology	6	6	413
3	Social Sciences	3	3	199
4	Engineering	1	1	25
5	Allergy	1	1	113

Mona M. Freidin

Publications

1.1K

Citations

H-Index

Page 1

	Year	Citations
Two Distinct Heterotypic Channels Mediate Gap Junction Coupling between Astrocyte and Oligodendrocyte Connexins Jennifer Orthmann‐Murphy, Mona M. Freidin, Esther Fischer, Journal of Neuroscience	2007	183
Hereditary spastic paraplegia is a novel phenotype for GJA12/GJC2 mutations Jennifer Orthmann‐Murphy, Ettore Salsano, Charles K. Abrams, Brain Progressive SpasticityGap Junction PlaquesCx47/cx43 ChannelsMendelian DisorderNeurological Disorder	2008	154
Cytokine regulation of substance P expression in sympathetic neurons. Mona M. Freidin, John A. Kessler Proceedings of the National Academy of Sciences Immune RegulationImmunologyImmunologic MechanismCytokine BiologyImmune System	1991	144
Cultured sympathetic neurons synthesize and release the cytokine interleukin 1 beta. Mona M. Freidin, Michael V. L. Bennett, John A. Kessler Proceedings of the National Academy of Sciences Immune SystemsImmunologyImmunologic MechanismImmune SystemCellular Physiology	1992	113
Cytokine-induced programmed death of cultured sympathetic neurons John A. Kessler, W.H. Ludlam, Mona M. Freidin, Neuron InflammationMedicinePhysiologyCell DeathNervous System	1993	102
Functional alterations in gap junction channels formed by mutant forms of connexin 32: evidence for loss of function as a pathogenic mechanism in the X-linked form of Charcot-Marie-Tooth disease Charles K. Abrams, Mona M. Freidin, Vytas K. Verselis, Brain Research Gap Junction ChannelsConnexin 32Signal TransductionMolecular PhysiologyCharcot-marie-tooth Disease	2001	74
Pathogenesis of X-Linked Charcot-Marie-Tooth Disease: Differential Effects of Two Mutations in Connexin 32 Charles K. Abrams, Mona M. Freidin, Feliksas F. Bukauskas, Journal of Neuroscience	2003	63
Connexin 32 increases the proliferative response of Schwann cells to neuregulin-1 (Nrg1) Mona M. Freidin, Samantha Asche, Thaddeus A. Bargiello, Proceedings of the National Academy of Sciences Myelin Specific GenesImmunologySchwann CellsCell DeathProliferative Response	2009	39
AAV1.NT-3 gene therapy for X-linked Charcot–Marie–Tooth neuropathy type 1 Burçak Özeş, Morgan Myers, Kyle Moss, Gene Therapy	2021	38
Properties of human connexin 31, which is implicated in hereditary dermatological disease and deafness Charles K. Abrams, Mona M. Freidin, Vytas K. Verselis, Proceedings of the National Academy of Sciences	2006	34

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