James B. Koprich - Concepedia

Author

James B. Koprich

University Health Network ORCID

Also Known As

J. Koprich, J.B. Koprich, James B Koprich, James B. Koprich, James Benjamin Koprich, James Koprich, Koprich, James B

Publications

4.1K

Citations

H-Index

Concepts

All Affiliations

University Health Network Toronto Western Hospital Krembil Research Institute Atuka (Canada)Universitätsklinikum Würzburg

About

James B. Koprich is an author at University Health Network specializing in medicine, neuroscience, and neuropharmacology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	18	21	1.3K
2	Social Sciences	11	11	874
3	Cell Biology	4	4	208
4	Health Sciences	1	1	28
5	Behavioral Sciences	1	1	62

James B. Koprich

Publications

3.2K

Citations

H-Index

Page 1

	Year	Citations
Dynamic Changes in Presynaptic and Axonal Transport Proteins Combined with Striatal Neuroinflammation Precede Dopaminergic Neuronal Loss in a Rat Model of AAV α-Synucleinopathy Chee Yeun Chung, James B. Koprich, Hasan K. Siddiqi, Journal of Neuroscience Rabphilin 3ASynaptic TransmissionSignificant DopaminergicNeurotransmissionSynaptic Signaling	2009	379
Neuroinflammation mediated by IL-1β increases susceptibility of dopamine neurons to degeneration in an animal model of Parkinson's disease James B. Koprich, Casper Reske-Nielsen, Prabhakar Mithal, Journal of Neuroinflammation	2008	323
AAV1/2-induced overexpression of A53T-α-synuclein in the substantia nigra results in degeneration of the nigrostriatal system with Lewy-like pathology and motor impairment: a new mouse model for Parkinson’s disease Chi Wang Ip, Laura-Christin Klaus, Akua A. Karikari, Acta Neuropathologica Communications	2017	169
Expression of human A53T alpha-synuclein in the rat substantia nigra using a novel AAV1/2 vector produces a rapidly evolving pathology with protein aggregation, dystrophic neurite architecture and nigrostriatal degeneration with potential to model the pathology of Parkinson's disease James B. Koprich, Tom H. Johnston, M. Gabriela Reyes, Molecular Neurodegeneration	2010	144
Early-onset impairment of the ubiquitin-proteasome system in dopaminergic neurons caused by α-synuclein Chris McKinnon, Mitchell L. de Snoo, Elise Gondard, Acta Neuropathologica Communications	2020	132
Treatment with Trehalose Prevents Behavioral and Neurochemical Deficits Produced in an AAV α-Synuclein Rat Model of Parkinson’s Disease Qing He, James B. Koprich, Ying Wang, Molecular Neurobiology Neurodegenerative DiseasesNeurobiology Of DiseaseSynaptic SignalingCellular NeuroscienceMedicine	2015	124
Neurodegeneration by α-synuclein-specific T cells in AAV-A53T-α-synuclein Parkinson’s disease mice Akua A. Karikari, Rhonda L. McFleder, Eliana Ribechini, Brain Behavior and Immunity	2022	110
Progressive Neurodegeneration or Endogenous Compensation in an Animal Model of Parkinson's Disease Produced by Decreasing Doses of Alpha-Synuclein James B. Koprich, Tom H. Johnston, Philippe Huot, PLoS ONE	2011	107
Subthalamic nucleus deep brain stimulation is neuroprotective in the A53T α‐synuclein Parkinson's disease rat model Thomas Musacchio, Maike Rebenstorff, Felix Fluri, Annals of Neurology	2017	103
The Toll-Like Receptor-3 Agonist Polyinosinic:Polycytidylic Acid Triggers Nigrostriatal Dopaminergic Degeneration Michela Deleidi, Penelope J. Hallett, James B. Koprich, Journal of Neuroscience InflammationDa NeuronsMedicineSynaptic TransmissionImmunology	2010	101

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