John N. Stallone - Concepedia

Author

John N. Stallone

Also Known As

J N Stallone, J. N. Stallone, J.N. Stallone, JOHN N. STALLONE, Jariatu Stallone, John N Stallone, John N. Stallone, John Stallone

Publications

2.4K

Citations

H-Index

Concepts

All Affiliations

Texas A&M University Northeast Ohio Medical University Ohio University Universidad Nacional Autónoma de México University of Tennessee Health Science Center

About

John N. Stallone is an author at Texas A&M University specializing in physiology, medicine, and endocrinology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	21	25	1.3K
2	Public Health	4	4	114
3	Health Sciences	3	3	155
4	Education	2	2	64
5	Engineering	1	1	49

John N. Stallone

Publications

2.1K

Citations

H-Index

Page 1

	Year	Citations
Testosterone relaxes coronary arteries by opening the large-conductance, calcium-activated potassium channel Viju P. Deenadayalu, Richard E. White, John N. Stallone, American Journal of Physiology-Heart and Circulatory Physiology Coronary ArteriesCardiac MuscleCardiovascular DiseaseAtherosclerosisTestosterone Relaxes	2001	212
Testosterone causes direct relaxation of rat thoracic aorta. C E Costarella, John N. Stallone, Gregory W. Rutecki, Journal of Pharmacology and Experimental Therapeutics	1996	159
Gender differences in the expression of heat shock proteins: the effect of estrogen M. R. Voss, John N. Stallone, Min Li, American Journal of Physiology-Heart and Circulatory Physiology	2003	153
Testosterone-induced relaxation of rat aorta is androgen structure specific and involves K<sup>+</sup>channel activation Andrew Q. Ding, John N. Stallone Journal of Applied Physiology	2001	119
Activation of G protein-coupled estrogen receptor induces endothelium-independent relaxation of coronary artery smooth muscle Xuan Yu, Handong Ma, Scott A. Barman, American Journal of Physiology-Endocrinology and Metabolism Coronary ArteriesMolecular PharmacologyClassic Estrogen ReceptorsMolecular PhysiologyCardiovascular Disease	2011	97
Kallistatin is a potent new vasodilator. Julie Chao, John N. Stallone, Yu Mei Liang, Journal of Clinical Investigation HypertensionCardiovascular PharmacologyRat AortaPharmacotherapyBlood Pressure	1997	92
Sexual dimorphism in vasopressin-induced contraction of rat aorta John N. Stallone, Joan T. Crofton, Leonard Share American Journal of Physiology-Heart and Circulatory Physiology HypertensionEndocrine MechanismVascular AdaptationPhysiologyMale Aortas	1991	77
Estrogen replacement restores flow-induced vasodilation in coronary arterioles of aged and ovariectomized rats Amanda J. LeBlanc, Rafael Reyes, Lori S. Kang, American Journal of Physiology-Regulatory, Integrative and Comparative Physiology Vascular DiseaseNitric OxideGynecologyFlow-induced VasodilationOvarian Aging	2009	74
Exercise training enhances flow-induced vasodilation in skeletal muscle resistance arteries of aged rats: role of PGI<sub>2</sub> and nitric oxide Scott A. Spier, Michael D. Delp, John N. Stallone, American Journal of Physiology-Heart and Circulatory Physiology AgingNitric OxideFlow-induced VasodilationCardiovascular FunctionMuscle Physiology	2007	72
G protein-coupled estrogen receptor 1 mediates relaxation of coronary arteries via cAMP/PKA-dependent activation of MLCP Xuan Yu, Fen Li, John N. Stallone, American Journal of Physiology-Endocrinology and Metabolism Mediates RelaxationMolecular PharmacologyCell SignalingMolecular SignalingPorcine Coronary Arteries	2014	60

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