Peter J. Reddig - Concepedia

Author

Peter J. Reddig

University of North Carolina at Chapel Hill

Also Known As

Alahari, Suresh K., P J Reddig, P. Reddig, Peter J Reddig, Peter J. Reddig, Xu, Dong

Publications

1.1K

Citations

H-Index

Concepts

All Affiliations

University of North Carolina at Chapel Hill University of Wisconsin–Madison

About

Peter J. Reddig is an author at University of North Carolina at Chapel Hill specializing in cell biology, medicine, and cell signaling.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	6	6	348
2	Cell Biology	6	6	348
3	Immunology	3	3	275
4	Natural Sciences	1	1	14
5	Cellular Physiology	1	1	20

Peter J. Reddig

Publications

440

Citations

H-Index

Page 1

	Year	Citations
Transgenic mice overexpressing protein kinase Cdelta in the epidermis are resistant to skin tumor promotion by 12-O-tetradecanoylphorbol-13-acetate. Peter J. Reddig, N E Dreckschmidt, H Ahrens, PubMed ImmunologyDermatologyTransgenic MicePkcdelta ProteinTumor Biology	1999	156
Transgenic mice overexpressing protein kinase C epsilon in their epidermis exhibit reduced papilloma burden but enhanced carcinoma formation after tumor promotion. Peter J. Reddig, Nancy E. Dreckschmidt, Jun Zou, PubMed Carcinoma FormationImmunologyPathologyLine 215Dermatology	2000	99
The integrin‐binding protein Nischarin regulates cell migration by inhibiting PAK Suresh K. Alahari, Peter J. Reddig, R. L. Juliano The EMBO Journal	2004	92
Regulation of p21-activated Kinase-independent Rac1 Signal Transduction by Nischarin Peter J. Reddig, Dong Xu, R. L. Juliano Journal of Biological Chemistry Pak StrandSignal TransductionSignaling PathwayCell RegulationMolecular Regulation	2005	35
Protein kinase Cδ-mediated signal to ornithine decarboxylase induction is independent of skin tumor suppression Deric L. Wheeler, Peter J. Reddig, Nancy E. Dreckschmidt, Oncogene Signal TransductionSkin Tumor SuppressionCutaneous BiologyDecarboxylase InductionDermatology	2002	24
Overexpression of Protein Kinase C-ε in the Mouse Epidermis Leads to a Spontaneous Myeloproliferative-Like Disease Deric L. Wheeler, Peter J. Reddig, Kristin J. Ness, American Journal Of Pathology Skin DevelopmentImmunologyMouse Epidermis LeadsCell DeathPathology	2005	20
The Essential in Vivo Function of the Herpes Simplex Virus UL42 Protein Correlates with Its Ability to Stimulate the Viral DNA Polymerase in Vitro Peter J. Reddig, Lynn A. Grinstead, Steven Monahan, Virology Viral ReplicationMolecular VirologyNatural SciencesViral Polymerase MechanismPathogenesis	1994	14

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