Hagit Eldar-Finkelman

Author

Also Known As

Eldar-Finkelman, Hagit, H Eldar, H Eldar-Finkelman, H. Eldar, H. Eldar-Finkelman, HAGIT ELDAR, Hagit Eldar, Hagit Eldar-Finkelman, Zhu, Hongxin

118

Publications

12.2K

Citations

H-Index

108

Concepts

All Affiliations

Tel Aviv University Weizmann Institute of Science University of Washington University of Miami Technical University of Darmstadt

About

Hagit Eldar-Finkelman is an author at Tel Aviv University specializing in medicine, cell biology, and cell signaling.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	28	34	3.3K
2	Natural Sciences	13	13	1.1K
3	Health Sciences	5	5	949
4	Social Sciences	4	4	392
5	Allergy	1	1	85

Hagit Eldar-Finkelman

Publications

10.7K

Citations

H-Index

Page 1

	Year	Citations
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) Daniel J. Klionsky, Kotb Abdelmohsen, Akihisa Abe, Autophagy	2016	5.9K
GSK-3 Inhibitors: Preclinical and Clinical Focus on CNS Hagit Eldar-Finkelman, Ana Martı́nez Frontiers in Molecular Neuroscience PharmacotherapyMedicinal ChemistryNeurologyNeuroimmunologyInhibitory Activity	2011	338
Phosphorylation of insulin receptor substrate 1 by glycogen synthase kinase 3 impairs insulin action Hagit Eldar-Finkelman, Edwin G. Krebs Proceedings of the National Academy of Sciences Tyrosine KinaseInsulin ReceptorInsulin SignalingMetabolic SyndromeSignaling Pathway	1997	313
Rapid antidepressive-like activity of specific glycogen synthase kinase-3 inhibitor and its effect on β-catenin in mouse hippocampus Oksana Kaidanovich‐Beilin, Anat Milman, Abraham Weizman, Biological Psychiatry Molecular PharmacologyMolecular NeurosciencePsychiatryMedicinePsychotropic Medication	2004	273
Increased glycogen synthase kinase-3 activity in diabetes- and obesity-prone C57BL/6J mice. Hagit Eldar-Finkelman, S A Schreyer, Michi M. Shinohara, Diabetes Insulin SignalingObesityMetabolic SyndromeBody CompositionObesity-prone C57bl/6j Mice	1999	246
Leptin Induces Insulin-like Signaling That Antagonizes cAMP Elevation by Glucagon in Hepatocytes Allan Z. Zhao, Michi M. Shinohara, Huang Daming, Journal of Biological Chemistry Antagonizes Camp ElevationInsulin SignalingGastrointestinal Peptide HormoneMetabolic SyndromeInsulin-like Signaling	2000	240
Inhibition of Glycogen Synthase Kinase-3 Ameliorates β-Amyloid Pathology and Restores Lysosomal Acidification and Mammalian Target of Rapamycin Activity in the Alzheimer Disease Mouse Model Limor Avrahami, Dorit Farfara, Maya Shaham-Kol, Journal of Biological Chemistry	2012	219
Overexpression of mitogen-activated protein kinase kinase (MAPKK) and its mutants in NIH 3T3 cells. Evidence that MAPKK involvement in cellular proliferation is regulated by phosphorylation of serine residues in its kinase subdomains VII and VIII. Rony Seger, Dalia Seger, A A Reszka, Journal of Biological Chemistry GeneticsCell ProliferationNih 3T3Signaling PathwayCell Regulation	1994	218
Inactivation of Glycogen Synthase Kinase-3 by Epidermal Growth Factor Is Mediated by Mitogen-activated Protein Kinase/p90 Ribosomal Protein S6 Kinase Signaling Pathway in NIH/3T3 Cells Hagit Eldar-Finkelman, Rony Seger, Jackie R. Vandenheede, Journal of Biological Chemistry	1995	215
Increased glucose uptake promotes oxidative stress and PKC-δ activation in adipocytes of obese, insulin-resistant mice Ilana Talior, Merav Yarkoni, Nava Bashan, American Journal of Physiology-Endocrinology and Metabolism	2003	170

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