Nicolas Dumaz - Concepedia

Author

Nicolas Dumaz

Inserm ORCID

Also Known As

Dressick, Walter, N DUMAZ, N Dumaz, N. Dumaz, Nicolas DUMAZ, Nicolas Dumaz

129

Publications

3.9K

Citations

H-Index

Concepts

All Affiliations

Inserm Hôpital Saint-Louis Université Paris Cité Assistance Publique – Hôpitaux de Paris Sorbonne Paris Cité

About

Nicolas Dumaz is an author at Inserm specializing in medicine, cell biology, and tumor biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	16	19	1.4K
2	Cell Biology	14	15	1.1K
3	Natural Sciences	5	5	226
4	Cellular Physiology	5	5	213
5	Engineering	2	2	28

Nicolas Dumaz

Publications

2.9K

Citations

H-Index

Page 1

	Year	Citations
Serine15 phosphorylation stimulates p53 transactivation but does not directly influence interaction with HDM2 Nicolas Dumaz The EMBO Journal Signal TransductionCell RegulationMolecular PathwayP53 TransactivationMolecular Biology	1999	446
In Melanoma, <i>RAS</i> Mutations Are Accompanied by Switching Signaling from BRAF to CRAF and Disrupted Cyclic AMP Signaling Nicolas Dumaz, Robert Hayward, Jan Martin, Cancer Research	2006	307
Specific UV-induced mutation spectrum in the p53 gene of skin tumors from DNA-repair-deficient xeroderma pigmentosum patients. Nicolas Dumaz, C Drougard, Alain Sarasin, Proceedings of the National Academy of Sciences	1993	275
Protein Kinase A Blocks Raf-1 Activity by Stimulating 14-3-3 Binding and Blocking Raf-1 Interaction with Ras Nicolas Dumaz, Richard Marais Journal of Biological Chemistry	2003	253
The role of UV-B light in skin carcinogenesis through the analysis of p53 mutations in squamous cell carcinomas of hairless mice Nicolas Dumaz Carcinogenesis P53 MutationsMajor Hot SpotsSomatic VariantOncogenic AgentMedicine	1997	146
Protein kinase CK1 is a p53‐threonine 18 kinase which requires prior phosphorylation of serine 15 Nicolas Dumaz, Diane Milne, David W. Meek FEBS Letters	1999	140
Skin Tumors Induced by Sorafenib; Paradoxic RAS–RAF Pathway Activation and Oncogenic Mutations of <i>HRAS</i> , <i>TP53</i> , and <i>TGFBR1</i> J.-P. Arnault, Christine Mateus, Bernard Escudier, Clinical Cancer Research	2011	120
ERK and PDE4 cooperate to induce RAF isoform switching in melanoma Amélie Marquette, Jocelyne André, M. Bagot, Nature Structural & Molecular Biology Tumor BiologyMedicineMelanomaCell BiologySystems Biology	2011	89
p53 mutations in skin and internal tumors of xeroderma pigmentosum patients belonging to the complementation group C. Nicolas Dumaz, Christiane Drougard, Marie‐Françoise Avril, PubMed OncologyP53 MutationsYoung Xp ChildrenMedicinePhotocarcinogenesis	1998	88
Cyclic AMP Blocks Cell Growth through Raf-1-Dependent and Raf-1-Independent Mechanisms Nicolas Dumaz, Yvonne Light, Richard Marais Molecular and Cellular Biology Cell ProliferationCell CycleCell GrowthCellular CampCellular Physiology	2002	79

Page 1