Reiko Nagashima - Concepedia

Author

Reiko Nagashima

Setsunan University

Also Known As

R Nagashima, R. Nagashima, Reiko Nagashima

Publications

649

Citations

H-Index

Concepts

All Affiliations

Setsunan University Kanazawa University Tokyo Medical Center University of Tsukuba

About

Reiko Nagashima is an author at Setsunan University specializing in medicine, neuroscience, and pharmacology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	12	13	414
2	Cell Biology	6	6	232
3	Health Sciences	5	5	158
4	Social Sciences	5	5	97
5	Biochemistry	3	3	60

Reiko Nagashima

Publications

620

Citations

H-Index

Page 1

	Year	Citations
Decreased expression of CXXC4 promotes a malignant phenotype in renal cell carcinoma by activating Wnt signaling Takahiro Kojima, Toru Shimazui, Shiro Hinotsu, Oncogene UrologyGenitourinary CancerMalignant DiseaseMedicineKidney Research	2008	96
Nuclear factor-kappa B nuclear translocation in the cochlea of mice following acoustic overstimulation Masatsugu Masuda, Reiko Nagashima, Sho Kanzaki, Brain Research Developmental BiologyHealth SciencesAudiologyNoiseAuditory Physiology	2005	75
In vivo depletion of endogenous glutathione facilitates trimethyltin-induced neuronal damage in the dentate gyrus of mice by enhancing oxidative stress Masanori Yoneyama, Norito Nishiyama, Makoto Shuto, Neurochemistry International Vivo DepletionMedicineLipid PeroxidationPhysiologyNeuropharmacology	2007	49
Endogenous and Exogenous Glucocorticoids Prevent Trimethyltin From Causing Neuronal Degeneration of the Mouse Brain In Vivo: Involvement of Oxidative Stress Pathways Makoto Shuto, Kei Higuchi, Chie Sugiyama, Journal of Pharmacological Sciences	2009	43
Enhanced biosynthesis of glutathione in the spiral ganglion of the cochlea after in vivo treatment with dexamethasone in mice Reiko Nagashima, Kiyokazu Ogita Brain Research Enhanced BiosynthesisBiochemistryMedicinePhysiologyCochlear Physiology	2006	31
Activation of c-Jun N-Terminal Kinase Cascades Is Involved in Part of the Neuronal Degeneration Induced by Trimethyltin in Cortical Neurons of Mice Makoto Shuto, Keiichi Seko, Nobuyuki Kuramoto, Journal of Pharmacological Sciences	2009	30
In vivo acute treatment with trimethyltin chloride causes neuronal degeneration in the murine olfactory bulb and anterior olfactory nucleus by different cascades in each region Koichi Kawada, Masanori Yoneyama, Reiko Nagashima, Journal of Neuroscience Research	2008	30
In vivo treatment with the K+ channel blocker 4-aminopyridine protects against kainate-induced neuronal cell death through activation of NMDA receptors in murine hippocampus Kiyokazu Ogita, Hiroaki Okuda, Mami Watanabe, Neuropharmacology NeurophysiologyMedicineNmda ReceptorsNeuropharmacologyNeuroprotection	2005	30
Mechanism Underlying the Protective Effect of Tempol and Nω-Nitro-L-arginine Methyl Ester on Acoustic Injury: Possible Involvement of c-Jun N-Terminal Kinase Pathway and Connexin26 in the Cochlear Spiral Ligament Reiko Nagashima, Taro Yamaguchi, Hajime Tanaka, Journal of Pharmacological Sciences	2010	28
Indomethacin Ameliorates Trimethyltin-Induced Neuronal Damage <i>in Vivo</i> by Attenuating Oxidative Stress in the Dentate Gyrus of Mice Nguyen Quynh Huong, Yukary Nakamura, Nobuyuki Kuramoto, Biological and Pharmaceutical Bulletin Lipid PeroxidationCell DeathOrganotin TrimethyltinExperimental PharmacologySocial Sciences	2011	25

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