Claire Hidalgo-Curtis

Author

Also Known As

C Hidalgo-Curtis, C. Hidalgo-Curtis, Claire E Hidalgo-Curtis, Claire E. Hidalgo-Curtis, Claire Hidalgo-Curtis, Claire Hidalgo‐Curtis

Publications

2.5K

Citations

H-Index

Concepts

All Affiliations

Wessex Regional Genetics Laboratory University of Southampton University Hospital Heidelberg Heidelberg University Jena University Hospital

About

Claire Hidalgo-Curtis is an author at Wessex Regional Genetics Laboratory specializing in medicine, cell biology, and genetics.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	6	6	1.3K
2	Cell Biology	4	4	1.2K
3	Immunology	1	1	21
4	Molecular Biology	1	1	62
5	Molecular Genetics	1	1	62

Claire Hidalgo-Curtis

Publications

2.5K

Citations

H-Index

Page 1

	Year	Citations
Inactivating mutations of the histone methyltransferase gene EZH2 in myeloid disorders Thomas Ernst, Andrew Chase, Joannah Score, Nature Genetics Histone ModificationsChromatinChromatin RemodelingEpigenetic ChangeGenetics	2010	1.1K
Frequent CBL mutations associated with 11q acquired uniparental disomy in myeloproliferative neoplasms Francis Grand, Claire Hidalgo-Curtis, Thomas Ernst, Blood	2009	369
EZH2 mutational status predicts poor survival in myelofibrosis Paola Guglielmelli, Flavia Biamonte, Joannah Score, Blood	2011	253
Mutations affecting mRNA splicing define distinct clinical phenotypes and correlate with patient outcome in myelodysplastic syndromes Frédérik Damm, Olivier Kosmider, Véronique Gelsi‐Boyer, Blood	2012	230
Inactivation of polycomb repressive complex 2 components in myeloproliferative and myelodysplastic/myeloproliferative neoplasms Joannah Score, Claire Hidalgo-Curtis, Amy V. Jones, Blood	2011	171
Transcription factor mutations in myelodysplastic/myeloproliferative neoplasms Thomas Ernst, Andrew Chase, Katerina Zoi, Haematologica	2010	73
Analysis of genomic breakpoints in p190 and p210 BCR–ABL indicate distinct mechanisms of formation J Score, Marı́a José Calasanz, O. Ottman, Leukemia Genome InstabilityGeneticsBcr GeneMolecular BiologyGenomic Mechanism	2010	62
The t(1;9)(p34;q34) and t(8;12)(p11;q15) fuse pre‐mRNA processing proteins <i>SFPQ (PSF)</i> and <i>CPSF6</i> to <i>ABL</i> and <i>FGFR1</i> Claire Hidalgo-Curtis, Andrew Chase, Milton R. Drachenberg, Genes Chromosomes and Cancer	2008	56
Identification of <i>FOXP1</i> and <i>SNX2</i> as novel <i>ABL1</i> fusion partners in acute lymphoblastic leukaemia Thomas Ernst, Joannah Score, Michael W. Deininger, British Journal of Haematology	2011	44
Detection and molecular monitoring of FIP1L1-PDGFRA-positive disease by analysis of patient-specific genomic DNA fusion junctions J Score, Christoph Walz, Jelena Jovanović, Leukemia Genome InstabilityMolecular MonitoringMedicineDna ReplicationPathology	2008	36

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