Nicolas W.J. Schröder

Lipoteichoic Acid (LTA) of Streptococcus pneumoniaeand Staphylococcus aureus Activates Immune Cells via Toll-like Receptor (TLR)-2, Lipopolysaccharide-binding Protein (LBP), and CD14, whereas TLR-4 and MD-2 Are Not Involved

Nicolas W.J. Schröder, Siegfried Morath, Christian Alexander et al. · Journal of Biological Chemistry · 2003 · 636 citations · Full text

Life Sciences, Biochemistry, Immunology +17

LPS-binding protein protects mice from septic shock caused by LPS or gram-negative bacteria.

Norbert Lamping, Rabea Dettmer, Nicolas W.J. Schröder et al. · Journal of Clinical Investigation · 1998 · 361 citations · Full text

Veterinary Medicine, Immunology, Pathobiology +17

Toll-like receptor (TLR) polymorphisms in African children: Common <i>TLR-4</i> variants predispose to severe malaria

Frank P. Mockenhaupt, Jakob P. Cramer, Lutz Hamann et al. · Proceedings of the National Academy of Sciences · 2005 · 321 citations · Full text

Veterinary Medicine, Immunology, Infectious Diseases +17

Toll-like Receptor-2 Mediates Treponema Glycolipid and Lipoteichoic Acid-induced NF-κB Translocation

Bastian Opitz, Nicolas W.J. Schröder, Ingo Spreitzer et al. · Journal of Biological Chemistry · 2001 · 232 citations · Full text

Life Sciences, Immunology, Microbiology +17

Heterozygous Arg753Gln Polymorphism of Human TLR-2 Impairs Immune Activation by <i>Borrelia burgdorferi</i> and Protects from Late Stage Lyme Disease

Nicolas W.J. Schröder, Isabel Diterich, Antje Zinke et al. · The Journal of Immunology · 2005 · 189 citations · Full text

Veterinary Medicine, Immunology, Infectious Diseases +17