Laís Amorim Sacramento

Author

Also Known As

LAÍS A. SACRAMENTO, Lais A Sacramento, Lais A. Sacramento, Lais Sacramento, Laís A Sacramento, Laís A. Sacramento, Laís Amorim Sacramento, Laís Sacramento, Laís Silva Sacramento, Sacramento, Lais A.

Publications

405

Citations

H-Index

Concepts

All Affiliations

Universidade de São Paulo Universidade Federal de Sergipe University of Pennsylvania The Ohio State University Instituto Nacional de Ciência e Tecnologia de Doenças Tropicais

About

Laís Amorim Sacramento is an author at Universidade de São Paulo specializing in medicine, immunology, and immune regulation.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	7	7	189
2	Immunology	7	7	189
3	Cell Biology	4	4	101
4	Allergy	2	2	78
5	Orthopaedics	1	1	23

Laís Amorim Sacramento

Publications

373

Citations

H-Index

Page 1

	Year	Citations
Interleukin-27 (IL-27) Mediates Susceptibility to Visceral Leishmaniasis by Suppressing the IL-17–Neutrophil Response Gustavo F. S. Quirino, Manuela Sales Lima Nascimento, Marcela Davoli-Ferreira, Infection and Immunity Il-27 ProductionInnate Immune SystemImmunologyImmune RegulationInnate Immunity	2016	51
Toll-Like Receptor 9 Signaling in Dendritic Cells Regulates Neutrophil Recruitment to Inflammatory Foci following Leishmania infantum Infection Laís Amorim Sacramento, Silvia Cellone Trevelin, Manuela Sales Lima Nascimento, Infection and Immunity InflammationToll-like ReceptorsLeishmania Infantum InfectionToll-like Receptor 9Inflammatory Foci	2015	40
Toll-Like Receptor 2 Is Required for Inflammatory Process Development during Leishmania infantum Infection Laís Amorim Sacramento, Jéssica L. da Costa, Mikhael Haruo Fernandes de Lima, Frontiers in Microbiology	2017	34
Efficacy of Probiotics in Children and Adolescents With Atopic Dermatitis: A Randomized, Double-Blind, Placebo-Controlled Study Paula Danielle Santa Maria Albuquerque de Andrade, Jorgete Maria e Silva, Vanessa Carregaro, Frontiers in Nutrition	2022	29
Leishmania infantum Parasites Subvert the Host Inflammatory Response through the Adenosine A2A Receptor to Promote the Establishment of Infection Mikhael Haruo Fernandes de Lima, Laís Amorim Sacramento, Gustavo F. S. Quirino, Frontiers in Immunology	2017	27
Microbiota instruct IL-17A-producing innate lymphoid cells to promote skin inflammation in cutaneous leishmaniasis Tej Pratap Singh, Augusto M. Carvalho, Laís Amorim Sacramento, PLoS Pathogens Microbial PathogensInnate Lymphoid CellsImmunologyImmune RegulationCutaneous Leishmaniasis	2021	27
Expression of SOFAT by T- and B-lineage cells may contribute to bone loss Christian Jarry, Elizabeth Ferreira Martinez, Daiane Cristina Peruzzo, Molecular Medicine Reports B-lineage CellsImmunologyImmune RegulationPathologyBone Repair	2016	23
Therapeutic Treatment of Arthritic Mice with 15-Deoxy Δ<sup>12,14</sup>-Prostaglandin J<sub>2</sub>(15d-PGJ<sub>2</sub>) Ameliorates Disease through the Suppression of Th17 Cells and the Induction of CD4<sup>+</sup>CD25<sup>−</sup>FOXP3<sup>+</sup>Cells Vanessa Carregaro, Marcelo Henrique Napimoga, Raphael Sanches Peres, Mediators of Inflammation	2016	23
Nucleosides Present on Phlebotomine Saliva Induce Immunossuppression and Promote the Infection Establishment Vanessa Carregaro, José M. C. Ribeiro, Jesús G. Valenzuela, PLoS neglected tropical diseases	2015	22
TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection Laís Amorim Sacramento, Luciana Benevides, Sandra Regina Maruyama, PLoS Pathogens Adaptive Immune SystemInnate Immune SystemImmunologyImmune RegulationPathology	2020	21

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