Szabolcs Fátrai - Concepedia

Concepedia

Author

Szabolcs Fátrai

University Medical Center Utrecht

Also Known As

S Fatrai, S. Fatrai, Szabolcs Fatrai, Szabolcs Fátrai

76

Publications

2.1K

Citations

20

H-Index

43

Concepts

All Affiliations

University Medical Center Utrecht Washington University in St. Louis University Medical Center Groningen University of Groningen University of Eastern Finland

SF

About

Szabolcs Fátrai is an author at University Medical Center Utrecht specializing in medicine, cell biology, and developmental biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	7	7	877
2	Cell Biology	6	6	735
3	Health Sciences	2	2	165
4	Diabetes	2	2	354
5	Cellular Physiology	1	1	199

Szabolcs Fátrai

×

21

Publications

2.1K

Citations

20

H-Index

	Year	Citations
Mice conditionally lacking the Wolfram gene in pancreatic islet beta cells exhibit diabetes as a result of enhanced endoplasmic reticulum stress and apoptosis A. C. Riggs, Ernesto Bernal‐Mizrachi, Mitsuru Ohsugi, Diabetologia Developmental BiologyDiabetesCell DeathDiabetes MellitusWolfram Gene	2005	226
Defective insulin secretion and increased susceptibility to experimental diabetes are induced by reduced Akt activity in pancreatic islet β cells Ernesto Bernal‐Mizrachi, Szabolcs Fátrai, James D. Johnson, Journal of Clinical Investigation	2004	212
Akt Induces β-Cell Proliferation by Regulating Cyclin D1, Cyclin D2, and p21 Levels and Cyclin-Dependent Kinase-4 Activity Szabolcs Fátrai, Lynda Elghazi, Norman Balcázar, Diabetes Cyclin D1ImmunologyCell ProliferationCell GrowthCellular Physiology	2006	199
SIRT1/PGC1α-Dependent Increase in Oxidative Phosphorylation Supports Chemotherapy Resistance of Colon Cancer Thomas T. Vellinga, Tijana Borovski, Vincent C. J. de Boer, Clinical Cancer Research	2015	193
Defective insulin secretion and increased susceptibility to experimental diabetes are induced by reduced Akt activity in pancreatic islet β cells Ernesto Bernal‐Mizrachi, Szabolcs Fátrai, James D. Johnson, Journal of Clinical Investigation	2004	188
Endoplasmic Reticulum Stress–Induced Apoptosis Is Partly Mediated by Reduced Insulin Signaling Through Phosphatidylinositol 3-Kinase/Akt and Increased Glycogen Synthase Kinase-3β in Mouse Insulinoma Cells Shanthi Srinivasan, Mitsuru Ohsugi, Zhonghao Liu, Diabetes	2005	161
Functional patient-derived organoid screenings identify MCLA-158 as a therapeutic EGFR × LGR5 bispecific antibody with efficacy in epithelial tumors Bram Herpers, Berina Eppink, Mark I. James, Nature Cancer MedicineImmunologyTumor ImmunityPathologyEpithelial Tumors	2022	142
Enhanced Polyamine Catabolism Alters Homeostatic Control of White Adipose Tissue Mass, Energy Expenditure, and Glucose Metabolism Eija Pirinen, Teemu Kuulasmaa, Marko Pietilä, Molecular and Cellular Biology Energy ExpenditureMetabolic RemodelingGlucose ToleranceInsulin SignalingOxidative Stress	2007	128
Collagen-rich stroma in aggressive colon tumors induces mesenchymal gene expression and tumor cell invasion Thomas T. Vellinga, S den Uil, Inne H. Borel Rinkes, Oncogene MedicinePathologyGastrointestinal Stromal TumorsColorectal CancerAggressive Colon Tumors	2016	108
GPx2 Suppression of H2O2 Stress Links the Formation of Differentiated Tumor Mass to Metastatic Capacity in Colorectal Cancer Benjamin L. Emmink, Jamila Laoukili, Anna P. Kipp, Cancer Research	2014	107

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