P. David Rogers - Concepedia

Author

P. David Rogers

University of Tennessee Health Science Center ORCID

Also Known As

, P David Rogers, P. D. Rogers, P. David Rogers, P.David Rogers, P. David Rogers, Paige Rogers, Phillip David Rogers, Phillip Rogers, Rogers, P.

139

Publications

8.7K

Citations

H-Index

Concepts

All Affiliations

University of Tennessee Health Science Center Le Bonheur Children's Hospital University of Würzburg Children's Medical Center University of Mississippi Medical Center

About

P. David Rogers is an author at University of Tennessee Health Science Center specializing in medicine, microbiology, and drug resistance.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	27	33	3K
2	Health Sciences	10	10	806
3	Natural Sciences	9	9	677
4	Engineering	4	4	450
5	Allergy	2	2	102

P. David Rogers

Publications

5.9K

Citations

H-Index

Page 1

	Year	Citations
The Transcription Factor Mrr1p Controls Expression of the MDR1 Efflux Pump and Mediates Multidrug Resistance in Candida albicans Joachim Morschhäuser, Katherine S. Barker, Teresa T. Liu, PLoS Pathogens	2007	347
Genome-Wide Expression Profiling of the Response to Azole, Polyene, Echinocandin, and Pyrimidine Antifungal Agents in<i>Candida albicans</i> Teresa T. Liu, Robin E. B. Lee, Katherine S. Barker, Antimicrobial Agents and Chemotherapy Antifungal AgentPyrimidine Antifungal AgentsGenome-wide Expression ProfilingPyrimidine BiosynthesisFungal Cell Biology	2005	311
Contribution of Clinically Derived Mutations in <i>ERG11</i> to Azole Resistance in Candida albicans Stephanie A. Flowers, Brendan Colón, Sarah Whaley, Antimicrobial Agents and Chemotherapy Clinically Derived MutationsAntifungal AgentAzole ResistanceAntifungal AgentsGenetics	2014	286
Mutations in the multi‐drug resistance regulator <i>MRR1</i>, followed by loss of heterozygosity, are the main cause of <i>MDR1</i> overexpression in fluconazole‐resistant <i>Candida albicans</i> strains Nico Dunkel, Julia Blaß, P. David Rogers, Molecular Microbiology Antifungal AgentHealth SciencesAntifungal AgentsCandida AlbicansMdr1 Gene	2008	277
Gain-of-Function Mutations in <i>UPC2</i> Are a Frequent Cause of <i>ERG11</i> Upregulation in Azole-Resistant Clinical Isolates of Candida albicans Stephanie A. Flowers, Katherine S. Barker, Elizabeth L. Berkow, Eukaryotic Cell	2012	235
A Gain-of-Function Mutation in the Transcription Factor Upc2p Causes Upregulation of Ergosterol Biosynthesis Genes and Increased Fluconazole Resistance in a Clinical <i>Candida albicans</i> Isolate Nico Dunkel, Teresa T. Liu, Katherine S. Barker, Eukaryotic Cell EngineeringFungal Cell BiologyGeneticsClinical MycologyMolecular Genetics	2008	233
Pdr1 regulates multidrug resistance in <i>Candida glabrata</i>: gene disruption and genome‐wide expression studies John‐Paul Vermitsky, Kelly D. Earhart, William Smith, Molecular Microbiology	2006	232
Genome-wide Expression Profiling of the Response to Polyene, Pyrimidine, Azole, and Echinocandin Antifungal Agents in Saccharomyces cerevisiae Ameeta K. Agarwal, P. David Rogers, Scott R. Baerson, Journal of Biological Chemistry Molecular BiologyPathologyDrug ResistanceBiosynthesisGenome-wide Expression Profiling	2003	208
Mutations in <i>TAC1B</i> : a Novel Genetic Determinant of Clinical Fluconazole Resistance in Candida auris Jeffrey M. Rybak, José F. Muñoz, Katherine S. Barker, mBio	2020	188
Genome-Wide Expression Profile Analysis Reveals Coordinately Regulated Genes Associated with Stepwise Acquisition of Azole Resistance in <i>Candida albicans</i> Clinical Isolates P. David Rogers, Katherine S. Barker Antimicrobial Agents and Chemotherapy	2003	155

Page 1