William J. Cain

The inverse association of plasma lipoprotein(a) concentrations with apolipoprotein(a) isoform size is not due to differences in Lp(a) catabolism but to differences in production rate.

Daniel J. Rader, William J. Cain, Katsunori Ikewaki et al. · Journal of Clinical Investigation · 1994 · 232 citations · Full text

Life Sciences, Biochemistry, Physiology +9

ABCA1 overexpression leads to hyperalphalipoproteinemia and increased biliary cholesterol excretion in transgenic mice

Boris Vaisman, Gilles Lambert, Marcelo Amar et al. · Journal of Clinical Investigation · 2001 · 216 citations · Full text

Life Sciences, Biochemistry, Molecular Biology +17

The low density lipoprotein receptor is not required for normal catabolism of Lp(a) in humans.

Daniel J. Rader, W. Alexander Mann, William J. Cain et al. · Journal of Clinical Investigation · 1995 · 206 citations · Full text

Life Sciences, Biochemistry, Biochemical Genetics +13

ABCA1 overexpression leads to hyperalphalipoproteinemia and increased biliary cholesterol excretion in transgenic mice

Boris Vaisman, Gilles Lambert, Marcelo Amar et al. · Journal of Clinical Investigation · 2001 · 206 citations · Full text

Life Sciences, Biochemistry, Molecular Biology +17

Variation in lipoprotein(a) concentrations among individuals with the same apolipoprotein (a) isoform is determined by the rate of lipoprotein(a) production.

Daniel J. Rader, William J. Cain, Loren A. Zech et al. · Journal of Clinical Investigation · 1993 · 192 citations · Full text

Life Sciences, Biochemistry, Biochemical Genetics +12