Kathryn E. Naus

The Absence of ABCA1 Decreases Soluble ApoE Levels but Does Not Diminish Amyloid Deposition in Two Murine Models of Alzheimer Disease

Veronica Hirsch‐Reinshagen, Luı́s F. Maia, Braydon L. Burgess et al. · Journal of Biological Chemistry · 2005 · 228 citations · Full text

Elevated plasma triglyceride levels precede amyloid deposition in Alzheimer’s disease mouse models with abundant Aβ in plasma

Braydon L. Burgess, Sean A. McIsaac, Kathryn E. Naus et al. · Neurobiology of Disease · 2006 · 124 citations · Full text

Neurodegenerative Diseases, Alzheimer's Disease, Amyloid Deposition +9

In vivo analysis of undocked connexin43 gap junction hemichannels in ovarian granulosa cells

Dan Tong, Tony Y. Li, Kathryn E. Naus et al. · Journal of Cell Science · 2007 · 63 citations

Molecular Biology, Cx43 Mutant, Cell Junctions +17

The cholesterol transporter ABCG1 modulates the subcellular distribution and proteolytic processing of β-amyloid precursor protein

Gavin Tansley, Braydon L. Burgess, Matt Bryan et al. · Journal of Lipid Research · 2007 · 52 citations · Full text

Subcellular Distribution, Alzheimer's Disease, β-Amyloid Precursor Protein +16

Overexpression of Human ABCG1 Does Not Affect Atherosclerosis in Fat-Fed ApoE-Deficient Mice

Braydon L. Burgess, Kathryn E. Naus, Jeniffer Chan et al. · Arteriosclerosis Thrombosis and Vascular Biology · 2008 · 52 citations