Wei Bin Fang - Concepedia

Author

Wei Bin Fang

University of Kansas Medical Center ORCID

Also Known As

Bruckheimer, Elizabeth, Fang Wei, Wei B. Fang, Wei Bin Fang, Wei Fang, Wei, Fang

Publications

2.4K

Citations

H-Index

Concepts

All Affiliations

University of Kansas Medical Center Vanderbilt University University of Kansas The University of Kansas Cancer Center Hubei University of Medicine

About

Wei Bin Fang is an author at University of Kansas Medical Center specializing in medicine, tumor biology, and cell biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	15	15	1.2K
2	Engineering	1	1	16
3	Health Sciences	1	1	30
4	Natural Sciences	1	1	151
5	Biochemistry	1	1	151

Wei Bin Fang

Publications

2.3K

Citations

H-Index

Page 1

	Year	Citations
The receptor tyrosine kinase EphA2 promotes mammary adenocarcinoma tumorigenesis and metastatic progression in mice by amplifying ErbB2 signaling Dana M. Brantley‐Sieders, Guanglei Zhuang, Donna J. Hicks, Journal of Clinical Investigation	2008	276
CCL2/CCR2 Chemokine Signaling Coordinates Survival and Motility of Breast Cancer Cells through Smad3 Protein- and p42/44 Mitogen-activated Protein Kinase (MAPK)-dependent Mechanisms Wei Bin Fang, Iman Jokar, An Zou, Journal of Biological Chemistry Tumor BiologyBreast OncologySignaling PathwayCell RegulationMedicine	2012	199
Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by BrafV600E and Pten Loss Lee Ann Jolly, Sergey V. Novitskiy, Philip Owens, Cancer Research Oncogenic BrafThyroid Cancer ProgressionMedicineThyroid DiseaseBiological Microenvironments	2016	156
Protein Phosphatase 2A Dephosphorylation of Phosphoserine 112 Plays the Gatekeeper Role for BAD-Mediated Apoptosis Chi-Wu Chiang, Cindy L. Kanies, Kwang Woon Kim, Molecular and Cellular Biology Bcl2 FamilyPhosphoserine 112ApoptosisCell DeathOxidative Stress	2003	151
Ephrin-A1 Facilitates Mammary Tumor Metastasis through an Angiogenesis-Dependent Mechanism Mediated by EphA Receptor and Vascular Endothelial Growth Factor in Mice Dana M. Brantley‐Sieders, Wei Bin Fang, Yoonha Hwang, Cancer Research	2006	143
Impaired tumor microenvironment in EphA2‐deficient mice inhibits tumor angiogenesis and metastatic progression Dana M. Brantley‐Sieders, Wei Bin Fang, Donna J. Hicks, The FASEB Journal Metastatic ProgressionReceptor TyrosineAngiogenesisEndothelial CellsImpaired Tumor Microenvironment	2005	129
Overexpression of EPHA2 receptor destabilizes adherens junctions via a RhoA-dependent mechanism Wei Bin Fang, Reneé C. Ireton, Guanglei Zhuang, Journal of Cell Science Src KinaseAdhesion DefectsCell AdhesionMolecular BiologyAdherens Junctions	2008	126
A kinase-dependent role for EphA2 receptor in promoting tumor growth and metastasis Wei Bin Fang, Dana M. Brantley‐Sieders, Monica A. Parker, Oncogene Signal TransductionKinase-dependent RoleSignaling PathwayEpha2 ReceptorTumor Growth	2005	125
Identification and Functional Analysis of Phosphorylated Tyrosine Residues within EphA2 Receptor Tyrosine Kinase Wei Bin Fang, Dana M. Brantley‐Sieders, Yoonha Hwang, Journal of Biological Chemistry	2008	124
Targeted gene silencing of CCL2 inhibits triple negative breast cancer progression by blocking cancer stem cell renewal and M2 macrophage recruitment Wei Bin Fang, Min Yao, Gage Brummer, Oncotarget	2016	115

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