Portia A. Vliet-Gregg

Author

University of Washington

Also Known As

Portia A. Vliet-Gregg, Portia Vliet-Gregg

Publications

334

Citations

H-Index

Concepts

All Affiliations

University of Washington Infectious Disease Research Institute Fred Hutch Cancer Center

About

Portia A. Vliet-Gregg is an author at University of Washington specializing in cell biology, medicine, and tumor biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	6	6	226
2	Cell Biology	6	6	226
3	Immunology	4	4	187
4	Natural Sciences	1	1	60
5	Molecular Biology	1	1	60

Portia A. Vliet-Gregg

Publications

281

Citations

H-Index

Page 1

	Year	Citations
NFX1-123 and Poly(A) Binding Proteins Synergistically Augment Activation of Telomerase in Human Papillomavirus Type 16 E6-Expressing Cells Rachel A. Katzenellenbogen, Erin Egelkrout, Portia A. Vliet-Gregg, Journal of Virology Cancer-associated VirusCell RegulationGeneticsHpv16 E6Immunology	2007	78
NFX1-123 Increases hTERT Expression and Telomerase Activity Posttranscriptionally in Human Papillomavirus Type 16 E6 Keratinocytes Rachel A. Katzenellenbogen, Portia A. Vliet-Gregg, Mei Xu, Journal of Virology Telomerase ActivityImmunologyPathologyMolecular BiologyHpv Type 16	2009	60
Cytoplasmic Poly(A) Binding Proteins Regulate Telomerase Activity and Cell Growth in Human Papillomavirus Type 16 E6-Expressing Keratinocytes Rachel A. Katzenellenbogen, Portia A. Vliet-Gregg, Mei Xu, Journal of Virology	2010	45
NFX1-123 and Human Papillomavirus 16E6 Increase Notch Expression in Keratinocytes Portia A. Vliet-Gregg, Jennifer Hamilton, Rachel A. Katzenellenbogen Journal of Virology ImmunologyCell DeathDermatologyTumor BiologyCancer-associated Virus	2013	36
Human papillomavirus 16E6 and NFX1-123 potentiate notch signaling and differentiation without activating cellular arrest Portia A. Vliet-Gregg, Jennifer Hamilton, Rachel A. Katzenellenbogen Virology Cellular ArrestCell RegulationHuman Papillomavirus VaccinesPathologyMedicine	2015	21
NFX1-123 is highly expressed in cervical cancer and increases growth and telomerase activity in HPV 16E6 expressing cells Portia A. Vliet-Gregg, Kristin Robinson, Justine Levan, Cancer Letters Tumor BiologyCancer-associated VirusCervical CancerMedicineTelomerase Activity	2019	18
Human papillomavirus type 16 E6 and NFX1-123 mislocalize immune signaling proteins and downregulate immune gene expression in keratinocytes Justine Levan, Portia A. Vliet-Gregg, Kristin Robinson, PLoS ONE Immune Gene ExpressionInnate Immune SystemImmunologyPathologyImmunologic Mechanism	2017	13
HPV type 16 E6 and NFX1–123 augment JNK signaling to mediate keratinocyte differentiation and L1 expression Justine Levan, Portia A. Vliet-Gregg, Kristin Robinson, Virology	2019	10

Page 1