Robert D. Gaffin - Concepedia

Author

Robert D. Gaffin

University of Illinois Chicago ORCID

Also Known As

Robert D. Gaffin, Robert Gaffin

Publications

609

Citations

H-Index

Concepts

All Affiliations

University of Illinois Chicago Cincinnati Children's Hospital Medical Center University of Cincinnati Medical Center Texas A&M University Texas A&M University System

About

Robert D. Gaffin is an author at University of Illinois Chicago specializing in medicine, physiology, and cardiovascular physiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	6	6	241
2	Engineering	2	2	48
3	Health Sciences	2	2	107
4	Applied Physiology	1	1	84
5	Biochemistry	1	1	23

Robert D. Gaffin

Publications

558

Citations

H-Index

Page 1

	Year	Citations
Enhancing natriuretic peptide signaling in adipose tissue, but not in muscle, protects against diet-induced obesity and insulin resistance Wei Wu, Fubiao Shi, Dianxin Liu, Science Signaling	2017	128
Inwardly rectifying K<sup>+</sup> channels are major contributors to flow‐induced vasodilatation in resistance arteries Sang Joon Ahn, Ibra S. Fancher, Jing‐Tan Bian, The Journal of Physiology	2016	100
Roles of phosphorylation of myosin binding protein‐C and troponin I in mouse cardiac muscle twitch dynamics Carl Tong, Robert D. Gaffin, David C. Zawieja, The Journal of Physiology Cardiac MuscleMuscle FunctionCytoskeletonMechanotransductionNormal Heart	2004	84
Desensitization of Myofilaments to Ca <sup>2+</sup> as a Therapeutic Target for Hypertrophic Cardiomyopathy With Mutations in Thin Filament Proteins Marco Stephan Lofrano‐Alves, Fernando Augusto Lavezzo Dias, Robert D. Gaffin, Circulation Cardiovascular Genetics	2014	67
Neonatal gene transfer of Serca2a delays onset of hypertrophic remodeling and improves function in familial hypertrophic cardiomyopathy James R. Peña, Ariani Cavazzani Szkudlarek, Chad M. Warren, Journal of Molecular and Cellular Cardiology Cardiac MuscleCardiomyopathyDevelopmental BiologyFamilial Hypertrophic CardiomyopathyGenetics	2010	49
Long-term rescue of a familial hypertrophic cardiomyopathy caused by a mutation in the thin filament protein, tropomyosin, via modulation of a calcium cycling protein Robert D. Gaffin, James R. Peña, Marco Stephan Lofrano‐Alves, Journal of Molecular and Cellular Cardiology Thin Filament ProteinCardiac MuscleCardiomyopathyHeart FailurePhysiology	2011	37
Arginine–glycine–aspartic acid (RGD)‐containing peptides inhibit the force production of mouse papillary muscle bundles via α<sub>5</sub>β<sub>1</sub> integrin Vandana Sarin, Robert D. Gaffin, Gerald A. Meininger, The Journal of Physiology Cardiac MuscleMuscle FunctionEngineeringArginine–glycine–aspartic AcidPeptide Science	2005	33
Charged residue changes in the carboxy‐terminus of α‐tropomyosin alter mouse cardiac muscle contractility Robert D. Gaffin, Kuppan Gokulan, James C. Sacchettini, The Journal of Physiology Cardiac MuscleMuscle FunctionCytoskeletonCellular PhysiologyMuscle Physiology	2004	23
Early sensitization of myofilaments to Ca2+ prevents genetically linked dilated cardiomyopathy in mice Marco Stephan Lofrano‐Alves, Chad M. Warren, Jillian N. Simon, Cardiovascular Research	2017	22
Changes in end-to-end interactions of tropomyosin affect mouse cardiac muscle dynamics Robert D. Gaffin, Kuppan Gokulan, James C. Sacchettini, American Journal of Physiology-Heart and Circulatory Physiology Cardiac MuscleMuscle FunctionEngineeringLeft Ventricular PressureCytoskeleton	2006	15

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