Nina Himmerkus - Concepedia

Author

Nina Himmerkus

Christian-Albrechts-Universität zu Kiel ORCID

Also Known As

Himmerkus, N., Himmerkus, Nina, N. Himmerkus, Nina HIMMERKUS, Nina Himmerkus

133

Publications

5.4K

Citations

H-Index

Concepts

All Affiliations

Christian-Albrechts-Universität zu Kiel Charité - Universitätsmedizin Berlin Max Delbrück Center Washington University in St. Louis Berlin Institute of Health at Charité - Universitätsmedizin Berlin

About

Nina Himmerkus is an author at Christian-Albrechts-Universität zu Kiel specializing in medicine, cellular physiology, and molecular physiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	14	15	1.3K
2	Natural Sciences	3	3	169
3	Biochemistry	2	2	108
4	Engineering	1	1	47
5	Social Sciences	1	1	48

Nina Himmerkus

Publications

5.1K

Citations

H-Index

Page 1

	Year	Citations
Synchronized renal tubular cell death involves ferroptosis Andreas Linkermann, Rachid Skouta, Nina Himmerkus, Proceedings of the National Academy of Sciences	2014	1.1K
Rip1 (Receptor-interacting protein kinase 1) mediates necroptosis and contributes to renal ischemia/reperfusion injury Andreas Linkermann, Jan Hinrich Bräsen, Nina Himmerkus, Kidney International Receptor-interacting ProteinApoptosisImmunologyRenal InflammationCell Death	2012	454
Acidified seawater impacts sea urchin larvae pH regulatory systems relevant for calcification Meike Stumpp, Marian Y. Hu, Frank Melzner, Proceedings of the National Academy of Sciences	2012	272
Claudin‐14 regulates renal Ca++ transport in response to CaSR signalling via a novel microRNA pathway Yongfeng Gong, Vijayaram Renigunta, Nina Himmerkus, The EMBO Journal	2012	248
Dysfunction of the key ferroptosis-surveilling systems hypersensitizes mice to tubular necrosis during acute kidney injury Wulf Tonnus, Claudia Meyer, Christian Steinebach, Nature Communications	2021	239
TRP4 (CCE1) Protein Is Part of Native Calcium Release-activated Ca2+-like Channels in Adrenal Cells Stephan Philipp, Claudia Trost, Jan Warnat, Journal of Biological Chemistry	2000	187
Deletion of <i>claudin-10</i> ( <i>Cldn10</i> ) in the thick ascending limb impairs paracellular sodium permeability and leads to hypermagnesemia and nephrocalcinosis Tilman Breiderhoff, Nina Himmerkus, Marchel Stuiver, Proceedings of the National Academy of Sciences Tight Junction ProteinsCellular PhysiologyParacellular PermeabilityRenal FunctionMembrane Transport	2012	155
Intrarenal Renin Angiotensin System Revisited Marcus Pohl, Henriette Josephine Kaminski, Hayo Castrop, Journal of Biological Chemistry HypertensionImmunologyRenal RasCytoskeletonCellular Physiology	2010	142
Hyperkalemic hypertension–associated cullin 3 promotes WNK signaling by degrading KLHL3 James A. McCormick, Chao-Ling Yang, Chong Zhang, Journal of Clinical Investigation	2014	138
The RIP1-Kinase Inhibitor Necrostatin-1 Prevents Osmotic Nephrosis and Contrast-Induced AKI in Mice Andreas Linkermann, Jan-Ole Heller, Ágnes Prókai, Journal of the American Society of Nephrology	2013	130

Page 1