Nathan C. Mitchell - Concepedia

Author

Nathan C. Mitchell

The University of Texas Health Science Center at San Antonio

Also Known As

N. C. Mitchell, N.C. Mitchell, Nathan C Mitchell, Nathan C. Mitchell, Nathan Clark Mitchell, Nathan Mitchell

Publications

312

Citations

H-Index

Concepts

All Affiliations

The University of Texas Health Science Center at San Antonio

About

Nathan C. Mitchell is an author at The University of Texas Health Science Center at San Antonio specializing in medicine, neuroscience, and social sciences.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	7	7	250
2	Social Sciences	6	6	232
3	Behavioral Sciences	1	1	20
4	Endocrinology	1	1	18
5	Neurology	1	1	21

Nathan C. Mitchell

Publications

285

Citations

H-Index

Page 1

	Year	Citations
Decynium-22 Enhances SSRI-Induced Antidepressant-Like Effects in Mice: Uncovering Novel Targets to Treat Depression Robert Horton, Deana M. Apple, W. A. Owens, Journal of Neuroscience Psychotropic MedicationPsychopharmacologyMental HealthSocial SciencesNovel Targets	2013	87
Revisiting Serotonin Reuptake Inhibitors and the Therapeutic Potential of “Uptake-2” in Psychiatric Disorders Lynette C. Daws, Wouter Koek, Nathan C. Mitchell ACS Chemical Neuroscience Psychiatric DisordersExtracellular SerotoninPsychotropic MedicationPsychopharmacologyAvailable Medications	2013	53
Antidepressant-like drug effects in juvenile and adolescent mice in the tail suspension test: Relationship with hippocampal serotonin and norepinephrine transporter expression and function Nathan C. Mitchell, Georgianna G. Gould, Corey M. Smolik, Frontiers in Pharmacology PsychopharmacologyHippocampal SerotoninTail Suspension TestPharmacotherapySocial Sciences	2013	29
Ontogeny of SERT Expression and Antidepressant-like Response to Escitalopram in Wild-Type and SERT Mutant Mice Nathan C. Mitchell, Georgianna G. Gould, Wouter Koek, Journal of Pharmacology and Experimental Therapeutics	2016	24
Activation of the hypothalamic paraventricular nucleus by acute intermittent hypoxia: Implications for sympathetic long-term facilitation neuroplasticity Nadia Oliveira Maruyama, Nathan C. Mitchell, Tamara Truong, Experimental Neurology Acute Intermittent HypoxiaAutonomic SystemHypothalamusNeuroanatomyNeurophysiology	2019	22
Neuroinflammation Contributes to High Salt Intake-Augmented Neuronal Activation and Active Coping Responses to Acute Stress T. Lee Gilman, Nathan C. Mitchell, Lynette C. Daws, The International Journal of Neuropsychopharmacology NeuropsychologyAffective NeurosciencePsychologySocial SciencesNeuroinflammation	2018	21
High Salt Intake Lowers Behavioral Inhibition T. Lee Gilman, Christina M. George, Mary Ann Andrade, Frontiers in Behavioral Neuroscience NeuropsychologySalt IntakeInhibitory ProcessAffective NeuroscienceSocial Sciences	2019	20
High salt intake enhances swim stress-induced PVN vasopressin cell activation and active stress coping Nathan C. Mitchell, T. Lee Gilman, Lynette C. Daws, Psychoneuroendocrinology Active StressOsmotic StressStress HormoneSodium HomeostasisStress	2018	18
Ontogeny of Norepinephrine Transporter Expression and Antidepressant-Like Response to Desipramine in Wild-Type and Serotonin Transporter Mutant Mice Nathan C. Mitchell, Melodi A. Bowman, Georgianna G. Gould, Journal of Pharmacology and Experimental Therapeutics	2016	11

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