Joseph J. Boyle - Concepedia

Author

Joseph J. Boyle

Imperial College London ORCID

Also Known As

J.J. BOYLE, Joseph Boyle, Joseph J Boyle, Joseph J. Boyle, Joseph James Boyle, Joseph J. Boyle

Publications

6.8K

Citations

H-Index

Concepts

All Affiliations

Imperial College London Hammersmith Hospital Lung Institute NIHR Imperial Biomedical Research Centre Addenbrooke's Hospital

About

Joseph J. Boyle is an author at Imperial College London specializing in medicine, inflammation, and immunology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	14	19	2.1K
2	Public Health	3	4	533
3	Engineering	1	1	13
4	Health Sciences	1	1	161
5	Natural Sciences	1	1	57

Joseph J. Boyle

Publications

6.3K

Citations

H-Index

Page 1

	Year	Citations
Cells of the adult human heart Monika Litviňuková, Carlos Talavera‐López, Henrike Maatz, Nature	2020	1.7K
Copy number polymorphism in Fcgr3 predisposes to glomerulonephritis in rats and humans Timothy J. Aitman, Rong Dong, Timothy J. Vyse, Nature Glomerular DiseaseGlomerulonephritisInflammationRenal FunctionDisease Mechanism	2006	678
Coronary Intraplaque Hemorrhage Evokes a Novel Atheroprotective Macrophage Phenotype Joseph J. Boyle, Heather A. Harrington, Emma Piper, American Journal Of Pathology InflammationThrombosisVascular DiseaseCardiovascular DiseaseImmunology	2009	308
Immunoglobulin M Is Required for Protection Against Atherosclerosis in Low-Density Lipoprotein Receptor–Deficient Mice Myles Lewis, Talat H. Malik, Michael R. Ehrenstein, Circulation	2009	246
Activating Transcription Factor 1 Directs Mhem Atheroprotective Macrophages Through Coordinated Iron Handling and Foam Cell Protection Joseph J. Boyle, Michael Johns, Theresa Kämpfer, Circulation Research	2011	212
Tumor Necrosis Factor-α Promotes Macrophage-Induced Vascular Smooth Muscle Cell Apoptosis by Direct and Autocrine Mechanisms Joseph J. Boyle, Peter L. Weissberg, Martin R. Bennett Arteriosclerosis Thrombosis and Vascular Biology	2003	192
Cooperative Interactions Between RB and p53 Regulate Cell Proliferation, Cell Senescence, and Apoptosis in Human Vascular Smooth Muscle Cells From Atherosclerotic Plaques Martin R. Bennett, Kirsty Macdonald, Shiu‐Wan Chan, Circulation Research	1998	191
EndoPDI, a Novel Protein-disulfide Isomerase-like Protein That Is Preferentially Expressed in Endothelial Cells Acts as a Stress Survival Factor D C Sullivan, John W. Moore, Joseph J. Boyle, Journal of Biological Chemistry	2003	176
Complement C1q Reduces Early Atherosclerosis in Low-Density Lipoprotein Receptor-Deficient Mice Vinay K. Bhatia, Sheng Yun, Viola Leung, American Journal Of Pathology InflammationMetabolic SyndromeComplement SystemVascular DiseaseCardiovascular Disease	2007	161
Human Blood-Derived Macrophages Induce Apoptosis in Human Plaque-Derived Vascular Smooth Muscle Cells by Fas-Ligand/Fas Interactions Joseph J. Boyle, David E. Bowyer, Peter L. Weissberg, Arteriosclerosis Thrombosis and Vascular Biology Vascular DiseaseApoptosisImmunologyCell DeathPathology	2001	140

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