Alpha-methylacyl-CoA racemase: a new molecular marker for prostate cancer.

Jun Luo, Shan Zha, Jessica Hicks, +8

PubMed

H2AX Prevents DNA Breaks from Progressing to Chromosome Breaks and Translocations

Sonia Franco, Monica Gostissa, Shan Zha, +13

Molecular Cell

ChromatinGenome InstabilityChromatin RemodelingGeneticsMolecular Biology +6

Cyclooxygenase-2 is up-regulated in proliferative inflammatory atrophy of the prostate, but not in prostate carcinoma.

Shan Zha, Wesley R. Gage, Jurga Sauvageot, +7

PubMed

Chemoprevention StrategyProliferative Inflammatory AtrophyPathologyCancer BiologyTumor Biology +14

Differential Phosphorylation of DNA-PKcs Regulates the Interplay between End-Processing and End-Ligation during Nonhomologous End-Joining

Wenxia Jiang, Jennifer Crowe, Xiangyu Liu, +9

Molecular Cell

ChromatinDna-pkcs RegulatesNatural SciencesEndocytic PathwayDna Replication +6

ATM damage response and XLF repair factor are functionally redundant in joining DNA breaks

Shan Zha, Chunguang Guo, Cristian Boboilă, +9

Nature

ChromatinGenome InstabilityDna DamageGeneticsXlf Repair Factor +8

Essential Role for DNA-PKcs in DNA Double-Strand Break Repair and Apoptosis in ATM-Deficient Lymphocytes

Elsa Callén, Mila Janković, Nancy Wong, +8

Molecular Cell

ChromatinGenome InstabilityDna DamageEssential RoleCell Regulation +10

The BCL11B tumor suppressor is mutated across the major molecular subtypes of T-cell acute lymphoblastic leukemia

Alejandro Gutiérrez, Alex Kentsis, Takaomi Sanda, +16

Blood

Lymphocyte-Specific Compensation for XLF/Cernunnos End-Joining Functions in V(D)J Recombination

Gang� Li, Frederick W. Alt, Hwei-Ling Cheng, +6

Molecular Cell

ImmunologyImmunologic MechanismImmune SystemMedicineCell Biology +1

XRCC1 prevents toxic PARP1 trapping during DNA base excision repair

Annie Albert Demin, Kouji Hirota, Masataka Tsuda, +11

Molecular Cell

Interactome analysis identifies a new paralogue of XRCC4 in non-homologous end joining DNA repair pathway

Mengtan Xing, Mingrui Yang, Wei Huo, +16

Nature Communications