Anja Krippner‐Heidenreich

Author

Also Known As

A Krippner-Heidenreich, A. Krippner-Heidenreich, A. Krippner‐Heidenreich, Anja KRIPPNER-HEIDENREICH, Anja Krippner-Heidenreich, Anja Krippner‐Heidenreich

Publications

1.4K

Citations

H-Index

Concepts

All Affiliations

Newcastle University University of Stuttgart Princess Máxima Center Max Delbrück Center Medizinische Hochschule Hannover

About

Anja Krippner‐Heidenreich is an author at Newcastle University specializing in medicine, cell biology, and cell signaling.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	14	15	709
2	Natural Sciences	4	4	226
3	Biochemistry	3	3	197
4	Engineering	2	2	72
5	Biomedical Engineering	2	2	72

Anja Krippner‐Heidenreich

Publications

1.3K

Citations

H-Index

Page 1

	Year	Citations
Targeting of the transcription factor Max during apoptosis: phosphorylation-regulated cleavage by caspase-5 at an unusual glutamic acid residue in position P1 Anja Krippner‐Heidenreich, Robert V. Talanian, Renate Sekul, Biochemical Journal Position P1ApoptosisCell DeathMyc/max/mad NetworkOxidative Stress	2001	108
Control of Receptor-induced Signaling Complex Formation by the Kinetics of Ligand/Receptor Interaction Anja Krippner‐Heidenreich, Fabian Tübing, Susanne Bryde, Journal of Biological Chemistry	2002	93
The Met-196 → Arg Variation of Human Tumor Necrosis Factor Receptor 2 (TNFR2) Affects TNF-α-induced Apoptosis by Impaired NF-κB Signaling and Target Gene Expression Andreas Till, Philip Rosenstiel, Anja Krippner‐Heidenreich, Journal of Biological Chemistry	2004	86
Dendritic Cell Maturation and Survival Are Differentially Regulated by TNFR1 and TNFR2 Nicola J. Maney, Gary Reynolds, Anja Krippner‐Heidenreich, The Journal of Immunology	2014	78
Tumor necrosis factor–costimulated T lymphocytes from patients with systemic sclerosis trigger collagen production in fibroblasts Thomas Hügle, Steven O’Reilly, Rachel E. Simpson, Arthritis & Rheumatism	2012	77
Targeting of the transcription factor Max during apoptosis: phosphorylation-regulated cleavage by caspase-5 at an unusual glutamic acid residue in position P1 Anja Krippner‐Heidenreich, Robert V. Talanian, Renate SEKUL, Biochemical Journal Position P1ApoptosisCell DeathTumor BiologyMyc/max/mad Network	2001	75
Potent antitumoral activity of TRAIL through generation of tumor-targeted single-chain fusion proteins B Schneider, Sabine Münkel, Anja Krippner‐Heidenreich, Cell Death and Disease ApoptosisImmunologyCell DeathImmunotherapyCancer Biology	2010	70
Single-Chain TNF, a TNF Derivative with Enhanced Stability and Antitumoral Activity Anja Krippner‐Heidenreich, Ingo Grunwald, Gudrun Zimmermann, The Journal of Immunology Antitumoral ActivitySingle-chain TnfImmunologyImmunologic MechanismTnf Monomers	2008	64
The Tumor Necrosis Factor Receptor Stalk Regions Define Responsiveness to Soluble versus Membrane-Bound Ligand C. Richter, Sylvia K. E. Messerschmidt, Gerlinde Holeiter, Molecular and Cellular Biology Proteinlipid InteractionSoluble TnfVersus Membrane-bound LigandImmunologyCell Death	2012	62
Inhibition of Proliferation and Apoptosis by the Transcriptional Repressor Mad1 Stefanie Gehring, Sabine Rottmann, Annette R. Menkel, Journal of Biological Chemistry	2000	55

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