Andrew D. Piekarz - Concepedia

Author

Andrew D. Piekarz

Indiana University – Purdue University Indianapolis

Also Known As

A. D. Piekarz, A. Piekarz, Andrew D Piekarz, Andrew D. Piekarz, Due, Michael R., Piekarz, Andrew D.

Publications

901

Citations

H-Index

Concepts

All Affiliations

Indiana University – Purdue University Indianapolis Indiana University School of Medicine Eli Lilly (United States)Neurosciences Institute

About

Andrew D. Piekarz is an author at Indiana University – Purdue University Indianapolis specializing in medicine, ion channels, and physiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	6	6	382
2	Social Sciences	4	4	310
3	Cell Biology	3	3	138
4	Health Sciences	1	1	50
5	Biochemistry	1	1	14

Andrew D. Piekarz

Publications

896

Citations

H-Index

Page 1

	Year	Citations
An Atypical Role for Collapsin Response Mediator Protein 2 (CRMP-2) in Neurotransmitter Release via Interaction with Presynaptic Voltage-gated Calcium Channels Joel M. Brittain, Andrew D. Piekarz, Yuying Wang, Journal of Biological Chemistry	2009	201
Human voltage-gated sodium channel mutations that cause inherited neuronal and muscle channelopathies increase resurgent sodium currents Brian W. Jarecki, Andrew D. Piekarz, James O. Jackson, Journal of Clinical Investigation Muscle ChannelopathiesMolecular PainNeurotransmissionSocial SciencesResurgent Sodium Currents	2010	143
Neuroexcitatory effects of morphine-3-glucuronide are dependent on Toll-like receptor 4 signaling Michael R. Due, Andrew D. Piekarz, Natalie M. Wilson, Journal of Neuroinflammation	2012	125
Insensitivity to pain induced by a potent selective closed-state Nav1.7 inhibitor M.L. Flinspach, Q. Xu, Andrew D. Piekarz, Scientific Reports	2017	121
Nav1.7 mutations associated with paroxysmal extreme pain disorder, but not erythromelalgia, enhance Navβ4 peptide‐mediated resurgent sodium currents Jonathan W. Theile, Brian W. Jarecki, Andrew D. Piekarz, The Journal of Physiology Pain DisordersPain MedicineSynaptic TransmissionMolecular BiologyNeuropathic Pain	2010	87
Tetrodotoxin-Resistant Sodium Channels in Sensory Neurons Generate Slow Resurgent Currents That Are Enhanced by Inflammatory Mediators Zhi‐Yong Tan, Andrew D. Piekarz, Birgit T. Priest, Journal of Neuroscience Synaptic TransmissionNeurotransmitterNeurotransmissionCellular NeurobiologyCellular Physiology	2014	66
CRMP-2 Peptide Mediated Decrease of High and Low Voltage-Activated Calcium Channels, Attenuation of Nociceptor Excitability, and Anti-Nociception in a Model of AIDS Therapy-Induced Painful Peripheral Neuropathy Andrew D. Piekarz, Michael R. Due, May Khanna, Molecular Pain	2012	56
Further insights into the antinociceptive potential of a peptide disrupting the N-type calcium channel–CRMP-2 signaling complex Sarah M. Wilson, Joel M. Brittain, Andrew D. Piekarz, Channels Pain MedicineSynaptic TransmissionFurther InsightsImmunologyNeuropathic Pain	2011	50
The human Nav1.5 F1486 deletion associated with long QT syndrome leads to impaired sodium channel inactivation and reduced lidocaine sensitivity Weihua Song, Yucheng Xiao, Hanying Chen, The Journal of Physiology F1486del ChannelsLong Qt SyndromeCardiac MuscleCellular PhysiologySodium Channel Inactivation	2012	22
Identification of the Benzyloxyphenyl Pharmacophore: A Structural Unit That Promotes Sodium Channel Slow Inactivation Amber M. King, Xiao-Fang Yang, Yuying Wang, ACS Chemical Neuroscience Synaptic TransmissionNeurotransmitterAnesthetic MechanismNeurotransmissionSocial Sciences	2012	14

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