Alberto Bosque - Concepedia

Author

Alberto Bosque

Also Known As

A Bosque, A Villalba Bosque, A. Bosque, Alberto Bosque, Benko, Erika, Bosque, Alberto, Linden, Noemi, Macedo, Amanda B., Singhania, Akul

121

Publications

4.6K

Citations

H-Index

Concepts

All Affiliations

University of Utah George Washington University Universidad de Zaragoza Cornell University University of North Carolina at Chapel Hill

About

Alberto Bosque is an author at University of Utah specializing in medicine, immunology, and hiv.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	19	21	1.3K
2	Cell Biology	16	16	950
3	Allergy	2	2	113
4	Engineering	1	1	28
5	Natural Sciences	1	1	84

Alberto Bosque

Publications

4.3K

Citations

H-Index

Page 1

	Year	Citations
An In-Depth Comparison of Latent HIV-1 Reactivation in Multiple Cell Model Systems and Resting CD4+ T Cells from Aviremic Patients Celsa A. Spina, Jenny L. Anderson, Nancie M. Archin, PLoS Pathogens	2013	414
Epigenetic Regulation of HIV-1 Latency by Cytosine Methylation Steven E. Kauder, Alberto Bosque, Annica Lindqvist, PLoS Pathogens	2009	366
Induction of HIV-1 latency and reactivation in primary memory CD4+ T cells Alberto Bosque, Vicente Planelles Blood	2008	341
Latent HIV reservoirs exhibit inherent resistance to elimination by CD8+ T cells Szu-Han Huang, Yanqin Ren, Allison S. Thomas, Journal of Clinical Investigation Adaptive Immune SystemMedicineHuman RetrovirusImmunologyLatent Hiv	2018	185
An HIV-Encoded Antisense Long Noncoding RNA Epigenetically Regulates Viral Transcription Sheena Saayman, Amanda Ackley, Anne‐Marie W. Turner, Molecular Therapy	2014	183
Homeostatic Proliferation Fails to Efficiently Reactivate HIV-1 Latently Infected Central Memory CD4+ T Cells Alberto Bosque, Marylinda Famiglietti, Andrew S. Weyrich, PLoS Pathogens Homeostatic ProliferationHomeostatic Proliferation FailsCentral Memory T-cellsAutoimmune DiseaseNeurovirology	2011	182
Apoptotic pathways are selectively activated by granzyme A and/or granzyme B in CTL-mediated target cell lysis Julián Pardo, Alberto Bosque, Reina Brehm, The Journal of Cell Biology	2004	163
BIRC2/cIAP1 Is a Negative Regulator of HIV-1 Transcription and Can Be Targeted by Smac Mimetics to Promote Reversal of Viral Latency Lars Pache, Míriam Santos Dutra, Adam M. Spivak, Cell Host & Microbe Viral LatencyHiv-1 TranscriptionHuman RetrovirusImmunologyAntiviral Therapy	2015	145
HIV-1 Vpr Triggers Natural Killer Cell–Mediated Lysis of Infected Cells through Activation of the ATR-Mediated DNA Damage Response Jeffrey P. Ward, Zachary Davis, Jason L. DeHart, PLoS Pathogens	2009	137
Reactivation of latent HIV-1 in central memory CD4+T cells through TLR-1/2 stimulation Camille L. Novis, Nancie M. Archin, María J. Buzón, Retrovirology	2013	129

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