Andrea J. Tenner - Concepedia

Author

Andrea J. Tenner

Also Known As

A J Tenner, A. J. Tenner, A. Tenner, A.J Tenner, A.J. Tenner, Andrea J. Tenner, Andrea J Tenner, Andrea J. Tenner, Andrea Joan Tenner, Andrea Tenner

264

Publications

18.2K

Citations

H-Index

172

Concepts

All Affiliations

University of California, Irvine Irvine University National Institute of Allergy and Infectious Diseases American Red Cross National Institutes of Health

About

Andrea J. Tenner is an author at University of California, Irvine specializing in medicine, immunology, and cell biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	48	66	6.7K
2	Natural Sciences	10	10	1K
3	Social Sciences	10	10	899
4	Health Sciences	6	6	493
5	Engineering	1	1	31

Andrea J. Tenner

129

Publications

14.5K

Citations

H-Index

Page 1

	Year	Citations
Adiponectin, a new member of the family of soluble defense collagens, negatively regulates the growth of myelomonocytic progenitors and the functions of macrophages Takafumi Yokota, Kenji Oritani, Isao Takahashi, Blood	2000	1.3K
Extensive innate immune gene activation accompanies brain aging, increasing vulnerability to cognitive decline and neurodegeneration: a microarray study David H. Cribbs, Nicole C. Berchtold, Victoria M. Perreau, Journal of Neuroinflammation	2012	495
Systematic phenotyping and characterization of the 5xFAD mouse model of Alzheimer’s disease Stefânia Forner, Shimako Kawauchi, Gabriela Balderrama-Gutierrez, Scientific Data	2021	422
A Dramatic Increase of C1q Protein in the CNS during Normal Aging Alexander Stephan, Daniel V. Madison, José Marı́a Mateos, Journal of Neuroscience C1q Protein LevelsSynapse EliminationC1q-deficient MiceImmunologyBiogerontology	2013	420
Absence of C1q Leads to Less Neuropathology in Transgenic Mouse Models of Alzheimer's Disease María Isabel Fonseca, Jun Zhou, Marina Botto, Journal of Neuroscience	2004	344
Cell-specific deletion of C1qa identifies microglia as the dominant source of C1q in mouse brain María Isabel Fonseca, Shu‐Hui Chu, Michael Hernandez, Journal of Neuroinflammation	2017	344
<i>beta</i>-Amyloid activates complement by binding to a specific region of the collagen-like domain of the C1q A chain. Hualiang Jiang, Debra Burdick, Charles Glabe, The Journal of Immunology	1994	334
Neuronal Protection in Stroke by an sLe <sup>x</sup> -Glycosylated Complement Inhibitory Protein Judy Huang, Louis J. Kim, Richard A. Mealey, Science	1999	314
Inflammatory Responses to Amyloidosis in a Transgenic Mouse Model of Alzheimer’s Disease Yasuji Matsuoka, Melanie Picciano, Brian Malester, American Journal Of Pathology InflammationInflammatory ResponsesNeurodegenerative DiseasesAlzheimer's DiseaseMedicine	2001	304
Human Pulmonary Surfactant Protein (SP-A), a Protein Structurally Homologous to C1q, Can Enhance FcR- and CR1-mediated Phagocytosis Andrea J. Tenner, Susan L. Robinson, J D Borchelt, Journal of Biological Chemistry Pulmonary SurfactantInflammatory Lung DiseaseLung InflammationImmunologyPulmonary Alveolar Proteinosis	1989	293

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