David M. Loeb - Concepedia

Author

David M. Loeb

Also Known As

D M Loeb, D. Loeb, D. M. Loeb, D.M. Loeb, David Loeb, David M Loeb, David M. Loeb, David Mark Loeb, Loeb,David, Zheng, Deyou

271

Publications

6.1K

Citations

H-Index

140

Concepts

All Affiliations

Johns Hopkins University Sidney Kimmel Comprehensive Cancer Center Johns Hopkins Medicine Albert Einstein College of Medicine Children's Hospital at Montefiore

About

David M. Loeb is an author at Johns Hopkins University specializing in medicine, pathology, and cell biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	27	39	2.5K
2	Health Sciences	10	10	386
3	Natural Sciences	3	3	355
4	Engineering	1	1	14
5	Allergy	1	1	24

David M. Loeb

Publications

5.1K

Citations

H-Index

Page 1

	Year	Citations
Trk receptors use redundant signal transduction pathways involving SHC and PLC-γ1 to mediate NGF responses Robert M. Stephens, David M. Loeb, Terry D. Copeland, Neuron	1994	540
The trk proto-oncogene rescues NGF responsiveness in mutant NGF-nonresponsive PC12 cell lines David M. Loeb, Johnna Maragos, Dionisio Martín‐Zanca, Cell Signal TransductionCell RegulationCell DivisionNatural SciencesCell Interaction	1991	252
Loss of cyclin D2 expression in the majority of breast cancers is associated with promoter hypermethylation. Ella Evron, Christopher B. Umbricht, Dorian Korz, PubMed Tumor BiologyBreast OncologyOncologyMedicinePromoter Hypermethylation	2001	213
Wilms' tumor suppressor gene (WT1) is expressed in primary breast tumors despite tumor-specific promoter methylation. David M. Loeb, Ella Evron, Chirag B. Patel, PubMed Primary Breast TumorsBreast OncologyEpigenetic ChangeGeneticsCancer Biology	2001	211
Use of a reconstituted basement membrane to measure cell invasiveness and select for highly invasive tumor cells. Victor P. Terranova, Erkki Hujanen, David M. Loeb, Proceedings of the National Academy of Sciences Biological MicroenvironmentsCellular PhysiologyTumor BiologyMatrix BiologyCell Invasiveness	1986	172
High ALDH Activity Identifies Chemotherapy-Resistant Ewing's Sarcoma Stem Cells That Retain Sensitivity to EWS-FLI1 Inhibition Ola Awad, Jason T. Yustein, Preeti Shah, PLoS ONE	2010	149
A Trk nerve growth factor (NGF) receptor point mutation affecting interaction with phospholipase C-gamma 1 abolishes NGF-promoted peripherin induction but not neurite outgrowth. David M. Loeb, Robert M. Stephens, T D Copeland, Journal of Biological Chemistry Normal Ngf BindingReceptor Point MutationPeripheral NervesCellular PhysiologyTrk Y785f	1994	132
Phase 2 study of dasatinib in patients with alveolar soft part sarcoma, chondrosarcoma, chordoma, epithelioid sarcoma, or solitary fibrous tumor Scott M. Schuetze, Vanessa Bolejack, Edwin Choy, Cancer	2016	128
Generation of chordoma cell line JHC7 and the identification of Brachyury as a novel molecular target Wesley Hsu, Ahmed Mohyeldin, Sagar Shah, Journal of neurosurgery	2011	125
IGF1R- and ROR1-Specific CAR T Cells as a Potential Therapy for High Risk Sarcomas Xin Huang, Haein Park, J.T. Greene, PLoS ONE High Risk SarcomasImmunologyImmunoeditingPathologyImmunotherapeutics	2015	106

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