Michael B. Reed - Concepedia

Author

Michael B. Reed

McGill University Health Centre ORCID

Also Known As

M. B. Reed, MICHAEL REED, Michael B Reed, Michael B. Reed, Michael Reed, Michael B. Reed

Publications

5.4K

Citations

H-Index

Concepts

All Affiliations

McGill University Health Centre McGill University National Institute of Allergy and Infectious Diseases National Institutes of Health The University of Melbourne

About

Michael B. Reed is an author at McGill University Health Centre specializing in medicine, pathogenesis, and tuberculosis.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	16	17	3.6K
2	Health Sciences	5	5	1.4K
3	Engineering	1	1	52
4	Natural Sciences	1	1	20
5	Biology	1	1	20

Michael B. Reed

Publications

5.3K

Citations

H-Index

Page 1

	Year	Citations
Pgh1 modulates sensitivity and resistance to multiple antimalarials in Plasmodium falciparum Michael B. Reed, Kevin J. Saliba, Sonia R. Caruana, Nature Systems BiologyPlasmodium FalciparumAntiparasitic AgentGeneticsPathogenesis	2000	857
A glycolipid of hypervirulent tuberculosis strains that inhibits the innate immune response Michael B. Reed, Pilar Domenech, Claudia Manca, Nature Host ResponseMedical MicrobiologyPulmonary TuberculosisPathogenesisImmunology	2004	746
DnaE2 Polymerase Contributes to In Vivo Survival and the Emergence of Drug Resistance in Mycobacterium tuberculosis Helena I. Boshoff, Michael B. Reed, Clifton E. Barry, Cell Pulmonary TuberculosisAntimicrobial Resistance GeneHealth SciencesMycobacterium TuberculosisMolecular Biology	2003	424
Contribution of the<i>Mycobacterium tuberculosis</i>MmpL Protein Family to Virulence and Drug Resistance Pilar Domenech, Michael B. Reed, Clifton E. Barry Infection and Immunity Tuberculosis PreventionImmunologyBacteriologyMolecular BiologyBacterial Pathogens	2005	354
Virulence of Selected<i>Mycobacterium tuberculosis</i>Clinical Isolates in the Rabbit Model of Meningitis Is Dependent on Phenolic Glycolipid Produced by the Bacilli Liana Tsenova, Evette Ellison, Ryhor Harbacheuski, The Journal of Infectious Diseases Pathogenic MicrobiologyMicrobial PathogensHumoral ResponseImmunologyPathology	2005	258
The W-Beijing Lineage of <i>Mycobacterium tuberculosis</i> Overproduces Triglycerides and Has the DosR Dormancy Regulon Constitutively Upregulated Michael B. Reed, Sébastien Gagneux, Kathryn DeRiemer, Journal of Bacteriology Tuberculosis PreventionBacteriologyImmunologyMolecular BiologyKlebsiella Pneumoniae	2007	237
Increased NOD2-mediated recognition of <i>N</i>-glycolyl muramyl dipeptide François Coulombe, Maziar Divangahi, Frédéric J. Veyrier, The Journal of Experimental Medicine	2009	224
Differential Monocyte Activation Underlies Strain-Specific<i>Mycobacterium tuberculosis</i>Pathogenesis Claudia Manca, Michael B. Reed, Sherry Freeman, Infection and Immunity InflammationVitro InfectionCytokinePulmonary TuberculosisMedical Microbiology	2004	220
Targeted disruption of an erythrocyte binding antigen in <i>Plasmodium falciparum</i> is associated with a switch toward a sialic acid-independent pathway of invasion Michael B. Reed, Sonia R. Caruana, Adrian H. Batchelor, Proceedings of the National Academy of Sciences	2000	197
Major <i>Mycobacterium tuberculosis</i> Lineages Associate with Patient Country of Origin Michael B. Reed, Victoria K. Pichler, Fiona McIntosh, Journal of Clinical Microbiology	2009	182

Page 1