Simon L. Currie - Concepedia

Author

Simon L. Currie

Howard Hughes Medical Institute ORCID

Also Known As

Currie, Simon L, Simon L Currie, Simon L. Currie

Publications

710

Citations

H-Index

Concepts

All Affiliations

Howard Hughes Medical Institute Huntsman Cancer Institute University of Utah The University of Texas Southwestern Medical Center Canada's Michael Smith Genome Sciences Centre

About

Simon L. Currie is an author at Howard Hughes Medical Institute specializing in natural sciences, medicine, and molecular biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	2	2	82
2	Natural Sciences	2	2	82
3	Cell Biology	2	2	82
4	Molecular Biology	2	2	82
5	Molecular Genetics	1	1	41

Simon L. Currie

Publications

658

Citations

H-Index

Page 1

	Year	Citations
Intrinsically disordered sequences enable modulation of protein phase separation through distributed tyrosine motifs Yuan Lin, Simon L. Currie, Michael K. Rosen Journal of Biological Chemistry	2017	442
Quantitative reconstitution of yeast RNA processing bodies Simon L. Currie, Wenmin Xing, Denise Muhlrad, Proceedings of the National Academy of Sciences	2023	49
Synergy of aromatic residues and phosphoserines within the intrinsically disordered DNA-binding inhibitory elements of the Ets-1 transcription factor Geneviève Desjardins, Charles A. Meeker, Niraja Bhachech, Proceedings of the National Academy of Sciences	2014	42
Structured and disordered regions cooperatively mediate DNA-binding autoinhibition of ETS factors ETV1, ETV4 and ETV5 Simon L. Currie, Desmond Lau, Jedediah J. Doane, Nucleic Acids Research Molecular RegulationGeneticsGenomic MechanismMolecular BiologyMolecular Genetics	2017	41
ETV4 and AP1 Transcription Factors Form Multivalent Interactions with three Sites on the MED25 Activator-Interacting Domain Simon L. Currie, Jedediah J. Doane, Kathryn S. Evans, Journal of Molecular Biology Transcriptional RegulationNatural SciencesMolecular BiologyMed25 Activator-interacting DomainBiomolecular Interaction	2017	41
Electrostatic repulsion causes anticooperative DNA binding between tumor suppressor ETS transcription factors and JUN–FOS at composite DNA sites Bethany J. Madison, Kathleen A. Clark, Niraja Bhachech, Journal of Biological Chemistry	2018	17
Interaction of the Jhd2 Histone H3 Lys-4 Demethylase with Chromatin Is Controlled by Histone H2A Surfaces and Restricted by H2B Ubiquitination Fu Huang, Saravanan Ramakrishnan, Srijana Pokhrel, Journal of Biological Chemistry	2015	14
Molecular features driving condensate formation and gene expression by the BRD4-NUT fusion oncoprotein are overlapping but distinct Martyna Kosno, Simon L. Currie, Ashwani Kumar, Scientific Reports	2023	12

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