Boris A. Leibovitch - Concepedia

Author

Boris A. Leibovitch

Icahn School of Medicine at Mount Sinai ORCID

Also Known As

B. A. LEIBOVITCH, B. A. Leibovitch, B.A. LEIBOVITCH, B.A. Leibovitch, Boris A Leibovitch, Boris A. Leibovitch, Boris Leibovitch

Publications

1.6K

Citations

H-Index

Concepts

All Affiliations

Icahn School of Medicine at Mount Sinai Institute of Cancer Research Institute of Molecular Genetics Sylvester Comprehensive Cancer Center Kurchatov Institute

About

Boris A. Leibovitch is an author at Icahn School of Medicine at Mount Sinai specializing in medicine, natural sciences, and molecular genetics.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	11	12	948
2	Natural Sciences	7	7	856
3	Biology	4	4	96
4	Health Sciences	1	1	34
5	Anesthesia	1	1	42

Boris A. Leibovitch

Publications

1.3K

Citations

H-Index

Page 1

	Year	Citations
Heterochromatic Silencing and HP1 Localization in <i>Drosophila</i> Are Dependent on the RNAi Machinery Manika Pal‐Bhadra, Boris A. Leibovitch, Sumit G. Gandhi, Science Epigenetic ChangeGeneticsPosition Effect VariegationGenomic MechanismMolecular Biology	2004	660
The MMSET protein is a histone methyltransferase with characteristics of a transcriptional corepressor Jotin Marango, Manabu Shimoyama, Hitomi Nishio, Blood	2007	199
Selective Inhibition of SIN3 Corepressor with Avermectins as a Novel Therapeutic Strategy in Triple-Negative Breast Cancer Yeon-Jin Kwon, Kevin Petrie, Boris A. Leibovitch, Molecular Cancer Therapeutics Peptide SciencePharmacotherapyCancer BiologyTamoxifen SensitivityTumor Biology	2015	84
Interference with Sin3 function induces epigenetic reprogramming and differentiation in breast cancer cells Eduardo Farias, Kevin Petrie, Boris A. Leibovitch, Proceedings of the National Academy of Sciences	2010	67
GAGA Factor and the TFIID Complex Collaborate in Generating an Open Chromatin Structure at the<i>Drosophila melanogaster hsp26</i>Promoter Boris A. Leibovitch, Quinn Lu, Lawrence R. Benjamin, Molecular and Cellular Biology	2002	57
Mutations That Affect Ion Channels Change the Sensitivity of<i>Drosophila Melanogaster</i>to Volatile Anesthetics Boris A. Leibovitch, K.S. Krishnan, H A Nash Journal of Neurogenetics Anesthetic MechanismMolecular GeneticsNeurotransmissionSensory SystemsMolecular Adaptation	1995	42
Targeting the SIN3A-PF1 interaction inhibits epithelial to mesenchymal transition and maintenance of a stem cell phenotype in triple negative breast cancer Nidhi Bansal, Kevin Petrie, Rossitza Christova, Oncotarget Sin3a-pf1 InteractionCancer BiologyTumor BiologyTranscriptional RegulationSignaling Pathway	2015	34
Influence of deficiency of the histone gene-containing 38B-40 region on X-chromosome template activity and the White gene position effect variegation in Drosophila melanogaster R. B. Khesin, Boris A. Leibovitch Molecular and General Genetics MGG BiologyChromatinDrosophila MelanogasterChromatin RemodelingNatural Sciences	1978	27
Invasive phenotype in triple negative breast cancer is inhibited by blocking SIN3A–PF1 interaction through KLF9 mediated repression of ITGA6 and ITGB1 Rama Kadamb, Boris A. Leibovitch, Eduardo Farias, Translational Oncology Sin3 Interaction DomainCancer BiologyTumor BiologyTranscriptional RegulationSin3a–pf1 Interaction	2021	24
Targeted interference of SIN3A-TGIF1 function by SID decoy treatment inhibits Wnt signaling and invasion in triple negative breast cancer cells Yeon-Jin Kwon, Boris A. Leibovitch, Nidhi Bansal, Oncotarget	2016	18

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