Masha Fridkis‐Hareli

Author

Harvard University ORCID

Also Known As

M Fridkis-Hareli, M. FRIDKIS‐HARELI, M. Fridkis-Hareli, M. Fridkis‐Hareli, Masha Fridkis-Hareli, Masha Fridkis‐Hareli

Publications

Citations

H-Index

Concepts

All Affiliations

Harvard University Weizmann Institute of Science Dana-Farber Cancer Institute Alexion Pharmaceuticals (United States)Los Alamos National Laboratory

About

Masha Fridkis‐Hareli is an author at Harvard University specializing in medicine, immunology, and autoimmunity.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	19	26	1.3K
2	Public Health	2	2	32
3	Education	1	1	15
4	Engineering	1	1	112
5	Natural Sciences	1	1	19

Masha Fridkis‐Hareli

Publications

1.8K

Citations

H-Index

Page 1

	Year	Citations
Direct binding of myelin basic protein and synthetic copolymer 1 to class II major histocompatibility complex molecules on living antigen-presenting cells--specificity and promiscuity. Masha Fridkis‐Hareli, Dvora Teitelbaum, E Gurevich, Proceedings of the National Academy of Sciences HistocompatibilityMhc BindingImmunologyImmune RegulationMolecular Biology	1994	245
Copolymer 1 inhibits chronic relapsing experimental allergic encephalomyelitis induced by proteolipid protein (PLP) peptides in mice and interferes with PLP-specific T cell responses Dvora Teitelbaum, Masha Fridkis‐Hareli, Ruth Arnon, Journal of Neuroimmunology InflammationAllergyInflammatory Lung DiseaseAutoimmune DiseaseCopolymer 1	1996	154
The complement receptor 2/factor H fusion protein TT30 protects paroxysmal nocturnal hemoglobinuria erythrocytes from complement-mediated hemolysis and C3 fragment opsonization Antonio M. Risitano, Rosario Notaro, Caterina Pascariello, Blood	2012	119
Design and development of TT30, a novel C3d-targeted C3/C5 convertase inhibitor for treatment of human complement alternative pathway–mediated diseases Masha Fridkis‐Hareli, Michael Storek, István Mazsaroff, Blood	2011	117
Promiscuous Binding of Synthetic Copolymer 1 to Purified HLA-DR Molecules Masha Fridkis‐Hareli, Jack L. Strominger The Journal of Immunology EngineeringImmunologyMolecular BiologyAntigen ProcessingImmunotherapy	1998	112
The autoimmune reactivity to myelin oligodendrocyte glycoprotein (MOG) in multiple sclerosis is potentially pathogenic: Effect of copolymer 1 on MOG-induced disease Avraham Ben‐Nun, Itzhack Mendel, Ronit Bakimer, Journal of Neurology Autoimmune DiseaseNeurological DisorderMedicineCopolymer 1Immunology	1996	108
Are desmoglein autoantibodies essential for the immunopathogenesis of pemphigus vulgaris, or just ‘witnesses of disease'? Ralf Paus, Masayuki Amagai, A. Razzaque Ahmed, Experimental Dermatology ImmunodeficienciesImmunologyPathologyCytoskeletonDermatology	2006	95
Binding Motifs of Copolymer 1 to Multiple Sclerosis- and Rheumatoid Arthritis-Associated HLA-DR Molecules Masha Fridkis‐Hareli, John M. Neveu, Renée A. Robinson, The Journal of Immunology Multiple Sclerosis-ImmunologyMolecular BiologyAntigen ProcessingImmunotherapy	1999	58
Amelioration of proteolipid protein 139–151-induced encephalomyelitis in SJL mice by modified amino acid copolymers and their mechanisms Joel N. H. Stern, Zsolt Illés, Jayagopala Reddy, Proceedings of the National Academy of Sciences Proteinlipid InteractionImmunologyImmunotherapyNeuroinflammationInflammation	2004	47
Immunogenetic mechanisms for the coexistence of organ-specific and systemic autoimmune diseases Masha Fridkis‐Hareli Journal of Autoimmune Diseases	2008	46

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