Hartmut Halfter

Flt3 mutations from patients with acute myeloid leukemia induce transformation of 32D cells mediated by the Ras and STAT5 pathways

Masao Mizuki, R. Fenski, Hartmut Halfter et al. · Blood · 2000 · 581 citations

Medicine, Genetics, Molecular Biology +9

Mutations in SEPT9 cause hereditary neuralgic amyotrophy

Gregor Kuhlenbäumer, Mark Hannibal, Eva Nelis et al. · Nature Genetics · 2005 · 264 citations

Life Sciences, Genetics, Neuroscience +12

Tyrosine 785 is a major determinant of Trk‐‐substrate interaction.

Axel Obermeier, Hartmut Halfter, Karl‐Heinz Wiesmüller et al. · The EMBO Journal · 1993 · 186 citations · Full text

Life Sciences, Biochemistry, Molecular Biology +12

Delineating an oncostatin M-activated STAT3 signaling pathway that coordinates the expression of genes involved in cell cycle regulation and extracellular matrix deposition of MCF-7 cells

Fang Zhang, Cong Li, Hartmut Halfter et al. · Oncogene · 2003 · 136 citations

Life Sciences, Cell Biology, Molecular Biology +15

High-Throughput Analysis of Genome-Wide Receptor Tyrosine Kinase Expression in Human Cancers Identifies Potential Novel Drug Targets

Carsten Müller‐Tidow, Joachim Schwäble, Björn Steffen et al. · Clinical Cancer Research · 2004 · 119 citations

Medicine, Oncology, Molecular Oncology +8