Ana Cristina Roginski

Author

Universidade Federal do Rio Grande do Sul ORCID

Also Known As

A. C. Roginski, Ana Cristina Roginski

Publications

222

Citations

H-Index

Concepts

All Affiliations

Universidade Federal do Rio Grande do Sul Hospital de Clínicas de Porto Alegre Universidade Regional Integrada do Alto Uruguai e das Missões Universidade Estadual de Campinas (UNICAMP)

About

Ana Cristina Roginski is an author at Universidade Federal do Rio Grande do Sul specializing in medicine, physiology, and mitochondrial function.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	5	5	99
2	Metabolism	4	4	69
3	Biochemistry	3	3	52
4	Health Sciences	2	2	38
5	Neurology	2	2	47

Ana Cristina Roginski

Publications

191

Citations

H-Index

Page 1

	Year	Citations
Early effects of LPS-induced neuroinflammation on the rat hippocampal glycolytic pathway Adriana Fernanda K. Vizuete, Fernanda Telles Fróes, Marina Seady, Journal of Neuroinflammation	2022	69
Mitochondrial dysfunction, oxidative stress, ER stress and mitochondria-ER crosstalk alterations in a chemical rat model of Huntington's disease: Potential benefits of bezafibrate Morgana Brondani, Ana Cristina Roginski, Rafael Teixeira Ribeiro, Toxicology Letters Reductive StressAlzheimer's DiseaseMitochondrial FunctionMitochondrial DysfunctionPhysiology	2023	30
Disturbance of bioenergetics and calcium homeostasis provoked by metabolites accumulating in propionic acidemia in heart mitochondria of developing rats Ana Cristina Roginski, Alessandro Wajner, Cristiane Cecatto, Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease Energy MetabolismPropionic AcidemiaBiochemistryMitochondrial FunctionMedicine	2020	21
Differential Age-Dependent Mitochondrial Dysfunction, Oxidative Stress, and Apoptosis Induced by Neonatal Hypoxia-Ischemia in the Immature Rat Brain Felipe Kawa Odorcyk, Rafael Teixeira Ribeiro, Ana Cristina Roginski, Molecular Neurobiology Mitochondrial FunctionReperfusion InjuryPhysiologyHypoxia (Medicine)Immature Rat Brain	2021	17
Impairment of mitochondrial bioenergetics and permeability transition induction caused by major long-chain fatty acids accumulating in VLCAD deficiency in skeletal muscle as potential pathomechanisms of myopathy Cristiane Cecatto, Alexandre Umpierrez Amaral, Ana Cristina Roginski, Toxicology in Vitro Mitochondrial MyopathyMechanobiologyMuscle FunctionBiochemistryMitochondrial Function	2019	16
Experimental evidence that maleic acid markedly compromises glutamate oxidation through inhibition of glutamate dehydrogenase and α-ketoglutarate dehydrogenase activities in kidney of developing rats Ana Cristina Roginski, Cristiane Cecatto, Simone Magagnin Wajner, Molecular and Cellular Biochemistry Glutamate DehydrogenaseExperimental EvidenceBiochemistrySodium HomeostasisMedicine	2019	15
Uncovering Actions of Type 3 Deiodinase in the Metabolic Dysfunction-Associated Fatty Liver Disease (MAFLD) Rafael Aguiar Marschner, Ana Cristina Roginski, Rafael Teixeira Ribeiro, Cells	2023	13
Ethylmalonic acid impairs bioenergetics by disturbing succinate and glutamate oxidation and induces mitochondrial permeability transition pore opening in rat cerebellum Leonardo de Moura Alvorcem, Renata Lopes Britto, Cristiane Cecatto, Journal of Neurochemistry	2021	10

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