Christopher E. Helt - Concepedia

Author

Christopher E. Helt

University of Rochester

Also Known As

C. E. Helt, Christopher E. Helt, Christopher Helt

Publications

637

Citations

H-Index

Concepts

All Affiliations

University of Rochester

About

Christopher E. Helt is an author at University of Rochester specializing in medicine, cell biology, and oxidative stress.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	8	8	550
2	Cell Biology	7	7	533
3	Cellular Physiology	2	2	79
4	Health Sciences	1	1	33
5	Natural Sciences	1	1	208

Christopher E. Helt

Publications

550

Citations

H-Index

Page 1

	Year	Citations
Ataxia Telangiectasia Mutated (ATM) and ATM and Rad3-related Protein Exhibit Selective Target Specificities in Response to Different Forms of DNA Damage Christopher E. Helt, William A. Cliby, Peter C. Keng, Journal of Biological Chemistry Dna DamageGeneticsGenomic MechanismMolecular BiologyCell Death	2004	208
DNA damage induces downregulation of histone gene expression through the G1 checkpoint pathway Chuan Su, Guang Gao, Sandra M. Schneider, The EMBO Journal Histone ModificationsChromatinGenome InstabilityDna DamageChromatin Remodeling	2004	130
p53-Dependent Induction of p21Cip1/WAF1/Sdi1 Protects against Oxygen-Induced Toxicity Christopher E. Helt Toxicological Sciences P53 AbundanceP21 MrnaSupplemental OxygenMedicineOncogenic Agent	2001	80
The role of p21<sup>CIP1/WAF1</sup> in growth of epithelial cells exposed to hyperoxia Raymond C. Rancourt, Peter C. Keng, Christopher E. Helt, American Journal of Physiology-Lung Cellular and Molecular Physiology PathologyCell CycleCellular PhysiologyTumor BiologyOxidative Stress	2001	46
The Cdk and PCNA domains on p21<sup>Cip1</sup>both function to inhibit G<sub>1</sub>/S progression during hyperoxia Christopher E. Helt, Rhonda J. Staversky, Yi‐Jang Lee, American Journal of Physiology-Lung Cellular and Molecular Physiology Kinase InhibitorsCell CycleCancer BiologyCellular PhysiologyOxidative Stress	2004	33
p53 Modulates Radiation Sensitivity Independent of p21 Transcriptional Activation Dawn J. Mazzatti, Yi‐Jang Lee, Christopher E. Helt, American Journal of Clinical Oncology P53 StatusTumor BiologySystems BiologyRadiation BiologyPhotocarcinogenesis	2005	20
The Rad9 protein enhances survival and promotes DNA repair following exposure to ionizing radiation Patrick D. Brandt, Christopher E. Helt, Peter C. Keng, Biochemical and Biophysical Research Communications Genome InstabilityDna DamageRadiation BiologyRadiation EffectRadiation Exposure	2006	17
p21Cip1/Waf1/Sdi1 protects against hyperoxia by maintaining expression of Bcl-XL Rhonda J. Staversky, Peter F. Vitiello, Sean C. Gehen, Free Radical Biology and Medicine Redox SignalingP21cip1/waf1/sdi1 ProtectsRedox BiologyMedicineCell Biology	2006	16

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