Colin L. Willis - Concepedia

Author

Colin L. Willis

Queen's Medical Centre

Also Known As

C. L. Willis, C. Willis, C.L WILLIS, C.L. Willis, Colin L Willis, Colin L. Willis, Colin Willis

Publications

1.1K

Citations

H-Index

Concepts

All Affiliations

Queen's Medical Centre University of New England University of Nottingham University of California, Irvine University of Leicester

About

Colin L. Willis is an author at Queen's Medical Centre specializing in medicine, neuroprotection, and neuroscience.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	10	10	482
2	Cell Biology	4	4	268
3	Social Sciences	3	3	65
4	Biochemistry	3	3	137
5	Natural Sciences	1	1	24

Colin L. Willis

Publications

Citations

H-Index

Page 1

	Year	Citations
Focal astrocyte loss is followed by microvascular damage, with subsequent repair of the blood‐brain barrier in the apparent absence of direct astrocytic contact Colin L. Willis, Christopher C. Nolan, Sharon N. Reith, Glia	2003	124
Reversible disruption of tight junction complexes in the rat blood‐brain barrier, following transitory focal astrocyte loss Colin L. Willis, Lopa Leach, Garry Clarke, Glia	2004	119
Protein Kinase C Activation Modulates Reversible Increase in Cortical Blood–Brain Barrier Permeability and Tight Junction Protein Expression during Hypoxia and Posthypoxic Reoxygenation Colin L. Willis, Diana Meske, Thomas P. Davis Journal of Cerebral Blood Flow & Metabolism	2010	109
Microglial activation and increased synthesis of complement component C1q precedes blood–brain barrier dysfunction in rats Nicholas J. Lynch, Colin L. Willis, Christopher C. Nolan, Molecular Immunology InflammationComplement SystemMicroglial ActivationBlood–brain BarrierMedicine	2003	100
A size selective vascular barrier in the rat area postrema formed by perivascular macrophages and the extracellular matrix Colin L. Willis, Claire J. Garwood, David Ray Neuroscience InflammationAngiogenesisVascular AdaptationRat Area PostremaPhysiology	2007	96
Occludin oligomeric assemblies at tight junctions of the blood–brain barrier are altered by hypoxia and reoxygenation stress Gwen McCaffrey, Colin L. Willis, William D. Staatz, Journal of Neurochemistry Low OxygenOccludin Oligomeric AssembliesReoxygenation StressOligomeric AssembliesRedox Biology	2009	92
Glia-Induced Reversible Disruption of Blood–Brain Barrier Integrity and Neuropathological Response of the Neurovascular Unit Colin L. Willis Toxicologic Pathology	2010	61
Protection Against N‐methyl‐D‐aspartate Receptor‐Mediated Neuronal Degeneration In Rat Brain by 7‐chlorokynurenate and 3‐amino‐1‐hydroxypyrrolid‐2‐one, Antagonists at The Allosteric Site for Glycine Alan C. Foster, Colin L. Willis, R. Tridgett European Journal of Neuroscience	1990	49
Partial recovery of the damaged rat blood–brain barrier is mediated by adherens junction complexes, extracellular matrix remodeling and macrophage infiltration following focal astrocyte loss Colin L. Willis, Ryan B. Camire, Stephanie A. Brule, Neuroscience Blood–brain BarrierMedicineBrain-immune InteractionBrain InjuryNeurology	2013	42
Transitory loss of glia and the subsequent modulation in inflammatory cytokines/chemokines regulate paracellular claudin-5 expression in endothelial cells Ryan B. Camire, Holly J. Beaulac, Colin L. Willis Journal of Neuroimmunology InflammationCytokineSignal TransductionEndothelial CellsTransitory Loss	2015	38

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