Nathalie Jurisch‐Yaksi

Author

Norwegian University of Science and Technology ORCID

Also Known As

Jurisch-Yaksi, Nathalie, Nathalie Jurisch-Yaksi, Nathalie Jurisch‐Yaksi

Publications

1.9K

Citations

H-Index

Concepts

All Affiliations

Norwegian University of Science and Technology St Olav's University Hospital University of Bonn German Center for Neurodegenerative Diseases KU Leuven

About

Nathalie Jurisch‐Yaksi is an author at Norwegian University of Science and Technology specializing in medicine, physiology, and neuroscience.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	8	8	320
2	Health Sciences	3	3	205
3	Natural Sciences	2	2	67
4	Social Sciences	2	2	121
5	Engineering	1	1	59

Nathalie Jurisch‐Yaksi

Publications

1.2K

Citations

H-Index

Page 1

	Year	Citations
Ciliary Beating Compartmentalizes Cerebrospinal Fluid Flow in the Brain and Regulates Ventricular Development Emilie Willoch Olstad, Christa Ringers, Jan N. Hansen, Current Biology	2019	224
Glia-neuron interactions underlie state transitions to generalized seizures Carmen Diaz Verdugo, Sverre Myren‐Svelstad, Ecem Aydin, Nature Communications	2019	153
Motile-Cilia-Mediated Flow Improves Sensitivity and Temporal Resolution of Olfactory Computations Ingrid Reiten, Fazil E. Uslu, Stephanie Fore, Current Biology BiologyNeurobiological MechanismNeural MechanismHealth SciencesNeurophysiology	2016	97
CiliaQ: a simple, open-source software for automated quantification of ciliary morphology and fluorescence in 2D, 3D, and 4D images Jan N. Hansen, Sebastian Rassmann, Birthe Stüven, The European Physical Journal E	2021	90
Diversity and function of motile ciliated cell types within ependymal lineages of the zebrafish brain Percival P. D’Gama, Tao Qiu, Mehmet İlyas Coşacak, Cell Reports	2021	85
Origin and role of the cerebrospinal fluid bidirectional flow in the central canal Olivier Thouvenin, Ludovic Keiser, Yasmine Cantaut-Belarif, eLife Cellular PhysiologyEpendymaCsf Flow BidirectionalityCerebrospinal FluidNeurology	2020	73
Junb regulates arterial contraction capacity, cellular contractility, and motility via its target Myl9 in mice Alexander H. Licht, Tobias Nübel, Anja Feldner, Journal of Clinical Investigation EngineeringMolecular RegulationCytoskeletonMechanotransductionCellular Physiology	2010	59
Loss of glutamate transporter <i>eaat2a</i> leads to aberrant neuronal excitability, recurrent epileptic seizures, and basal hypoactivity Adriana L. Hotz, Ahmed Jamali, Nicolas N Rieser, Glia	2021	57
Nanobody-directed targeting of optogenetic tools to study signaling in the primary cilium Jan N. Hansen, Fabian Kaiser, Christina Klausen, eLife Signal TransductionNanobody-directed TargetingSignaling PathwayCamp SignalingNanobiotechnology	2020	53
Extended structure–activity study of thienopyrimidine-based EGFR inhibitors with evaluation of drug-like properties Steffen Bugge, Audun Formo Buene, Nathalie Jurisch‐Yaksi, European Journal of Medicinal Chemistry Medicinal ChemistryPharmaceutical ScienceMedicineDrug DiscoveryRational Drug Design	2015	47

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