Karine Ancolio

Wild-type but Not Parkinson's Disease-related Ala-53 → Thr Mutant α-Synuclein Protects Neuronal Cells from Apoptotic Stimuli

Cristine Alvès da Costa, Karine Ancolio, Frédéric Checler · Journal of Biological Chemistry · 2000 · 211 citations · Full text

Life Sciences, Neurobiology, Neuroscience +17

Unusual phenotypic alteration of β amyloid precursor protein (βAPP) maturation by a new Val-715 → Met βAPP-770 mutation responsible for probable early-onset Alzheimer’s disease

Karine Ancolio, Cécile Dumanchin, Hélène Barelli et al. · Proceedings of the National Academy of Sciences · 1999 · 177 citations · Full text

Life Sciences, Genetics, Neuroscience +14

Characterization of New Polyclonal Antibodies Specific for 40 and 42 Amino Acid-Long Amyloid β Peptides: Their Use to Examine the Cell Biology of Presenilins and the Immunohistochemistry of Sporadic Alzheimer’s Disease and Cerebral Amyloid Angiopathy Cases

Hélène Barelli, Anthony Lebeau, Jean Vizzavona et al. · Molecular Medicine · 1997 · 140 citations · Full text

Life Sciences, Neurology, Neuroscience +17

Wild-type and mutated presenilins 2 trigger p53-dependent apoptosis and down-regulate presenilin 1 expression in HEK293 human cells and in murine neurons

Cristine Alvès da Costa, Erwan Paitel, Mark P. Mattson et al. · Proceedings of the National Academy of Sciences · 2002 · 132 citations

Life Sciences, Cell Biology, Neuroscience +17

α-Synuclein and the Parkinson's disease-related mutant Ala53Thr-α-synuclein do not undergo proteasomal degradation in HEK293 and neuronal cells

Karine Ancolio, Cristine Alvès da Costa, K. Uéda et al. · Neuroscience Letters · 2000 · 107 citations

Life Sciences, Molecular Biology, Neuroscience +15