Concepedia

Author

Mohamed Rahmani

Virginia Commonwealth UniversityORCID

Also Known As

Abdullah A. Toor, M Rahmani, M. Rahmani, Maha H. Philip Rahmani, Mohamed Rahmani, Mohammed Rahmani, Mohibullah Rahmani, Perier, Celine, RAHMANI, Mohammed

191

Publications

17K

Citations

54

H-Index

68

Concepts

All Affiliations

Virginia Commonwealth UniversityVirginia Commonwealth University Medical CenterInstitute of Cytochemistry and Molecular PharmacologyImmunovaccine (Canada)University of Sharjah
MR

About

Mohamed Rahmani is an author at Virginia Commonwealth University specializing in medicine, cell biology, and cell death.

Top Concepts

#ConceptH-IndexPublicationsCitations

1

Medicine

19

21

1.9K

2

Cell Biology

18

19

1.8K

3

Health Sciences

3

3

305

4

Biochemistry

3

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293

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Cellular Physiology

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Mohamed Rahmani

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78

Publications

16.3K

Citations

51

H-Index

YearCitations

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, Kotb Abdelmohsen, Akihisa Abe,

Autophagy

2016

5.9K

Global, regional, and national burden of diabetes from 1990 to 2021, with projections of prevalence to 2050: a systematic analysis for the Global Burden of Disease Study 2021

Kanyin Liane Ong, Lauryn K Stafford, Susan A. McLaughlin,

The Lancet

2023

3.7K

Apoptosis Induced by the Kinase Inhibitor BAY 43-9006 in Human Leukemia Cells Involves Down-regulation of Mcl-1 through Inhibition of Translation

Mohamed Rahmani, Eric Maynard Davis, Cheryl Bauer,

Journal of Biological Chemistry

2005

284

The proteasome inhibitor bortezomib interacts synergistically with histone deacetylase inhibitors to induce apoptosis in Bcr/Abl+ cells sensitive and resistant to STI571

Chunrong Yu, Mohamed Rahmani, Daniel H. Conrad,

Blood

2003

261

The Kinase Inhibitor Sorafenib Induces Cell Death through a Process Involving Induction of Endoplasmic Reticulum Stress

Mohamed Rahmani, Eric Maynard Davis, Timothy Ryan Crabtree,

Molecular and Cellular Biology

2007

248

A selective sphingosine kinase 1 inhibitor integrates multiple molecular therapeutic targets in human leukemia

Steven W. Paugh, Barbara S. Paugh, Mohamed Rahmani,

Blood

2008

246

Blockade of Histone Deacetylase Inhibitor-Induced RelA/p65 Acetylation and NF-κB Activation Potentiates Apoptosis in Leukemia Cells through a Process Mediated by Oxidative Damage, XIAP Downregulation, and c-Jun N-Terminal Kinase 1 Activation

Yun Dai, Mohamed Rahmani, Paul Dent,

Molecular and Cellular Biology

2005

241

A Bcr/Abl-independent, Lyn-dependent Form of Imatinib Mesylate (STI-571) Resistance Is Associated with Altered Expression of Bcl-2

Yun Dai, Mohamed Rahmani, Seth J. Corey,

Journal of Biological Chemistry

2004

236

Coadministration of Histone Deacetylase Inhibitors and Perifosine Synergistically Induces Apoptosis in Human Leukemia Cells through Akt and ERK1/2 Inactivation and the Generation of Ceramide and Reactive Oxygen Species

Mohamed Rahmani, Erin Reese, Yun Dai,

Cancer Research

2005

193

Pharmacologic mitogen-activated protein/extracellular signal-regulated kinase kinase/mitogen-activated protein kinase inhibitors interact synergistically with STI571 to induce apoptosis in Bcr/Abl-expressing human leukemia cells.

Chunrong Yu, Geoffrey W. Krystal, Lyuba Varticovksi,

PubMed

2002

192

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