Alper Yetil

Cellular senescence is an important mechanism of tumor regression upon c-Myc inactivation

Chi-Hwa Wu, Jan van Riggelen, Alper Yetil et al. · Proceedings of the National Academy of Sciences · 2007 · 413 citations · Full text

Pathology, Cell Proliferation, Cancer Biology +16

Resistance to Antiangiogenic Therapy Is Associated with an Immunosuppressive Tumor Microenvironment in Metastatic Renal Cell Carcinoma

Xian-De Liu, Anh Hoang, Lijun Zhou et al. · Cancer Immunology Research · 2015 · 211 citations · Full text

Urology, Cancer Immunosurveillance, Genitourinary Cancer +14

The interaction between Myc and Miz1 is required to antagonize TGFβ-dependent autocrine signaling during lymphoma formation and maintenance

Jan van Riggelen, Judith M. Müller, Tobias Otto et al. · Genes & Development · 2010 · 107 citations · Full text

Lymphoma Formation, Lymphoid Neoplasia, Signaling Pathway +15

Characterization of a synthetic human LINE-1 retrotransposon ORFeus-Hs

Wenfeng An, Lixin Dai, Anna Maria Niewiadomska et al. · Mobile DNA · 2011 · 69 citations · Full text

p19ARF is a critical mediator of both cellular senescence and an innate immune response associated with MYC inactivation in mouse model of acute leukemia

Alper Yetil, Benedict Anchang, Arvin M. Gouw et al. · Oncotarget · 2015 · 21 citations · Full text