Mohammed A. Matlib - Concepedia

Author

Mohammed A. Matlib

Also Known As

M A Matlib, M Abdul Matlib, M Matlib, M. A. MATLIB, M. A. Matlib, M. Abdul Matlib, M.A. MATLIB, M.A. Matlib, M.Abdul Matlib, Matlib

Publications

3.6K

Citations

H-Index

Concepts

All Affiliations

University of Cincinnati Medical Center University of Cincinnati

About

Mohammed A. Matlib is an author at University of Cincinnati Medical Center specializing in medicine, physiology, and pharmacology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	30	43	2.7K
2	Natural Sciences	7	7	406
3	Health Sciences	6	6	377
4	Public Health	4	4	173
5	Engineering	2	2	25

Mohammed A. Matlib

Publications

3.5K

Citations

H-Index

Page 1

	Year	Citations
Oxygen-bridged Dinuclear Ruthenium Amine Complex Specifically Inhibits Ca2+ Uptake into Mitochondria in Vitroand in Situ in Single Cardiac Myocytes Mohammed A. Matlib, Zhuan Zhou, Selena Knight, Journal of Biological Chemistry	1998	312
Defective intracellular Ca<sup>2+</sup> signaling contributes to cardiomyopathy in Type 1 diabetic rats Kin M. Choi, Yan Zhong, Brian D. Hoit, American Journal of Physiology-Heart and Circulatory Physiology Cardiac MuscleHeart FailureCardiovascular FunctionCellular PhysiologyType 1	2002	244
Selectivity of Inhibition of Na+-Ca2+ Exchange of Heart Mitochondria by Benzothiazepine CGP-37157 David A. Cox, Laura Conforti, Nicholas Sperelakis, Journal of Cardiovascular Pharmacology Cardiac MuscleHeart FailurePharmacotherapyBenzothiazepine Cgp-37157Experimental Pharmacology	1993	188
Mitochondrial dysfunction accompanies diastolic dysfunction in diabetic rat heart C. E. Flarsheim, I L Grupp, Mohammed A. Matlib American Journal of Physiology-Heart and Circulatory Physiology Cardiac MuscleHeart FailureDiabetic Rat HeartMetabolic RemodelingInsulin Signaling	1996	169
A role for the mitochondrial Na(+)-Ca2+ exchanger in the regulation of oxidative phosphorylation in isolated heart mitochondria. David Cox, Mohammed A. Matlib Journal of Biological Chemistry Cardiac MuscleMitochondrial BiologyRedox BiologyOxidative StressMolecular Physiology	1993	161
Specific binding of [3H]nitrendipine to membranes from coronary arteries and heart in relation to pharmacological effects. Paradoxical stimulation by diltiazem A. Depover, Mohammed A. Matlib, S.W. Lee, Biochemical and Biophysical Research Communications Coronary ArteriesMolecular PharmacologyParadoxical StimulationCardiovascular DiseaseMedicine	1982	158
Selective inhibition of Na+-induced Ca2+ release from heart mitochondria by diltiazem and certain other Ca2+ antagonist drugs. Pál L. Vághy, James D. Johnson, Mohammed A. Matlib, Journal of Biological Chemistry Cardiac MuscleCardiovascular PharmacologyCa2+ UptakePharmacotherapyExperimental Pharmacology	1982	151
Altered SR protein expression associated with contractile dysfunction in diabetic rat hearts Yan Zhong, Saadia Ahmed, Ingrid L. Grupp, American Journal of Physiology-Heart and Circulatory Physiology	2001	134
Compensatory Mechanisms Associated With the Hyperdynamic Function of Phospholamban-Deficient Mouse Hearts Guoxiang Chu, Wusheng Luo, Jay P. Slack, Circulation Research Cardiac MuscleHeart FailurePhospholamban-deficient Mouse HeartsCardiovascular FunctionCellular Physiology	1996	120
Effects of diltiazem, a calcium antagonist, on regional myocardial function and mitochondria after brief coronary occlusion Taku Nagao, Mohammed A. Matlib, Dean Franklin, Journal of Molecular and Cellular Cardiology Cardiovascular DiseaseCalcium AntagonistMedicinePhysiologyBrief Coronary Occlusion	1980	119

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