John A. Corbett - Concepedia

Author

John A. Corbett

Also Known As

Corbett, John Andrew, J A Corbett, J Corbett, J. A. CORBETT, J. A. Corbett, J. CORBETT, J. Corbett, J.A Corbett, J.A. Corbett, JOHN A. CORBETT

272

Publications

15.7K

Citations

H-Index

240

Concepts

All Affiliations

Saint Louis University Washington University in St. Louis Medical College of Wisconsin University of Alabama at Birmingham University of Birmingham

About

John A. Corbett is an author at Saint Louis University specializing in medicine, cell signaling, and immunology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	49	94	8.6K
2	Health Sciences	19	23	2.4K
3	Natural Sciences	5	5	976
4	Education	4	4	140
5	Arts	1	1	111

John A. Corbett

170

Publications

13.9K

Citations

H-Index

Page 1

	Year	Citations
Selective inhibition of the inducible nitric oxide synthase by aminoguanidine Thomas P. Misko, William M. Moore, Thomas P. Kasten, European Journal of Pharmacology Selective InhibitionMolecular PharmacologyBiochemistryReactive Nitrogen SpecieMedicine	1993	724
Aminoguanidine, a Novel Inhibitor of Nitric Oxide Formation, Prevents Diabetic Vascular Dysfunction John A. Corbett, Ronald G. Tilton, Kathy Chang, Diabetes	1992	609
Nitric oxide mediates cytokine-induced inhibition of insulin secretion by human islets of Langerhans. John A. Corbett, Michael A. Sweetland, Jaw‐Lin Wang, Proceedings of the National Academy of Sciences Nitric OxideImmunologyInsulin SignalingInflammationMetabolic Syndrome	1993	408
Expression of Neuregulins and their Putative Receptors, ErbB2 and ErbB3, Is Induced during Wallerian Degeneration Steven L. Carroll, Michele L. Miller, Paul W. Frohnert, Journal of Neuroscience Schwann Cell DedifferentiationPeripheral Nerve InjuryCell DeathPutative ReceptorsPeripheral Nerve	1997	328
Prevention of Diabetic Vascular Dysfunction by Guanidines: Inhibition of Nitric Oxide Synthase Versus Advanced Glycation End-Product Formation Ronald G. Tilton, Katherine Chang, Khalid S. Hasan, Diabetes	1993	288
Targeted Overactivity of β Cell KATP Channels Induces Profound Neonatal Diabetes Joseph C. Koster, Bess A. Marshall, Nancy J. Ensor, Cell Molecular PhysiologySignal TransductionPhysiologyDiabetesIon Channels	2000	288
Interleukin 1 beta induces the formation of nitric oxide by beta-cells purified from rodent islets of Langerhans. Evidence for the beta-cell as a source and site of action of nitric oxide. John A. Corbett, Jaw‐Lin Wang, Michael A. Sweetland, Journal of Clinical Investigation Nitric OxideImmunologyInsulin SignalingOxidative StressInflammation	1992	285
Interleukin-1 beta-induced formation of EPR-detectable iron-nitrosyl complexes in islets of Langerhans. Role of nitric oxide in interleukin-1 beta-induced inhibition of insulin secretion. John A. Corbett, Jack R. Lancaster, Michael A. Sweetland, Journal of Biological Chemistry Nitric OxideImmunologyInsulin SignalingOxidative StressInflammation	1991	259
Release of nitric oxide during the T cell-independent pathway of macrophage activation. Its role in resistance to Listeria monocytogenes Karen Beckerman, Howard W. Rogers, John A. Corbett, The Journal of Immunology	1993	250
IL-1.beta. induces the coexpression of both nitric oxide synthase and cyclooxygenase by Islets of Langerhans: activation of cyclooxygenase by nitric oxide John A. Corbett, Guim Kwon, John Turk, Biochemistry Pge2 ProductionNitric OxideImmunologyInsulin SignalingOxidative Stress	1993	250

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